Interplay between kinase domain autophosphorylation and F-actin binding domain in regulating imatinib sensitivity and nuclear import of BCR-ABL. (Q33828334)

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English	Interplay between kinase domain autophosphorylation and F-actin binding domain in regulating imatinib sensitivity and nuclear import of BCR-ABL.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

29 July 2017

Interplay between kinase domain autophosphorylation and F-actin binding domain in regulating imatinib sensitivity and nuclear import of BCR-ABL. (English)

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Europe PubMed Central

PubMed publication ID

29 July 2017

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based on heuristic

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Paolo Vigneri

2

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author name string

Martin Preyer

1

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Europe PubMed Central

PubMed publication ID

29 July 2017

Jean Y J Wang

3

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Europe PubMed Central

PubMed publication ID

29 July 2017

language of work or name

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publication date

11 February 2011

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Europe PubMed Central

PubMed publication ID

29 July 2017

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Europe PubMed Central

PubMed publication ID

29 July 2017

6

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Europe PubMed Central

PubMed publication ID

29 July 2017

2

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Europe PubMed Central

PubMed publication ID

29 July 2017

e17020

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Europe PubMed Central

PubMed publication ID

29 July 2017

copyright license

Creative Commons Attribution 4.0 International

11 February 2011

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April 2022 Public Data File from Crossref

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0 references

Orientation and oligomerization specificity of the Bcr coiled-coil oligomerization domain

1 reference

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28 July 2018

c-Abl regulation: a tail of two lipids

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Autoinhibition of Bcr-Abl through its SH3 domain

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Two distinct phosphorylation pathways have additive effects on Abl family kinase activation

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Structural basis for the autoinhibition of c-Abl tyrosine kinase

1 reference

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Phosphotyrosine mapping in Bcr/Abl oncoprotein using phosphotyrosine-specific immonium ion scanning

1 reference

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Profiling of tyrosine phosphorylation pathways in human cells using mass spectrometry

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Crystal structures of the kinase domain of c-Abl in complex with the small molecule inhibitors PD173955 and imatinib (STI-571)

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Autoinhibition of c-Abl

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Structure of the Bcr-Abl oncoprotein oligomerization domain

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1 reference

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1 reference

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Induction of apoptosis in chronic myelogenous leukemia cells through nuclear entrapment of BCR-ABL tyrosine kinase.

1 reference

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The NH(2)-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-Abl

1 reference

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Selection and characterization of BCR-ABL positive cell lines with differential sensitivity to the tyrosine kinase inhibitor STI571: diverse mechanisms of resistance.

1 reference

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Mechanism of resistance to the ABL tyrosine kinase inhibitor STI571 in BCR/ABL-transformed hematopoietic cell lines.

1 reference

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Induction of resistance to the Abelson inhibitor STI571 in human leukemic cells through gene amplification.

1 reference

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Leptomycin B inactivates CRM1/exportin 1 by covalent modification at a cysteine residue in the central conserved region

1 reference

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Polarized distribution of Bcr-Abl in migrating myeloid cells and co-localization of Bcr-Abl and its target proteins

1 reference

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Nuclear-cytoplasmic shuttling of C-ABL tyrosine kinase

1 reference

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CRM1 is responsible for intracellular transport mediated by the nuclear export signal

1 reference

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Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells

1 reference

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The cytostatic function of c-Abl is controlled by multiple nuclear localization signals and requires the p53 and Rb tumor suppressor gene products

1 reference

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An actin-binding function contributes to transformation by the Bcr-Abl oncoprotein of Philadelphia chromosome-positive human leukemias

1 reference

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Subcellular localization of Bcr, Abl, and Bcr-Abl proteins in normal and leukemic cells and correlation of expression with myeloid differentiation

1 reference

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Abrogation of retinoblastoma protein function by c-Abl through tyrosine kinase-dependent and -independent mechanisms

1 reference

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The mouse type IV c-abl gene product is a nuclear protein, and activation of transforming ability is associated with cytoplasmic localization

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Nonmyristoylated Abl proteins transform a factor-dependent hematopoietic cell line

1 reference

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Comparable complex rearrangements involving 8;21 and 9;22 translocations in leukaemia

1 reference

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A coiled-coil oligomerization domain of Bcr is essential for the transforming function of Bcr-Abl oncoproteins

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0017020

21 January 2018

The coiled-coil domain and Tyr177 of bcr are required to induce a murine chronic myelogenous leukemia-like disease by bcr/abl.

1 reference

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Exclusion of c-Abl from the Nucleus Restrains the p73 Tumor Suppression Function

1 reference

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A Src-like inactive conformation in the abl tyrosine kinase domain

1 reference

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Mechanisms of autoinhibition and STI-571/imatinib resistance revealed by mutagenesis of BCR-ABL.

1 reference

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Structural Basis for the Cytoskeletal Association of Bcr-Abl/c-Abl

1 reference

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NMR Assignment Reveals an α-Helical Fold for the F-Actin Binding Domain of Human Bcr-Abl/c-Abl

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0017020

21 January 2018

Fused transcript of abl and bcr genes in chronic myelogenous leukaemia

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0017020

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The tyrosine kinase inhibitor CGP57148B selectively inhibits the growth of BCR-ABL-positive cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/21347248

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A comparison of the activity, sequence specificity, and CRM1-dependence of different nuclear export signals

1 reference

https://pubmed.ncbi.nlm.nih.gov/21347248

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

c-Abl has high intrinsic tyrosine kinase activity that is stimulated by mutation of the Src homology 3 domain and by autophosphorylation at two distinct regulatory tyrosines

1 reference

https://pubmed.ncbi.nlm.nih.gov/21347248

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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10.1371/JOURNAL.PONE.0017020

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

2011PLoSO...617020P

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PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

ResearchGate publication ID

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