Inhibition of hyaluronan synthesis protects against central nervous system (CNS) autoimmunity and increases CXCL12 expression in the inflamed CNS. (Q34044654)

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English	Inhibition of hyaluronan synthesis protects against central nervous system (CNS) autoimmunity and increases CXCL12 expression in the inflamed CNS.	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

Inhibition of hyaluronan synthesis protects against central nervous system (CNS) autoimmunity and increases CXCL12 expression in the inflamed CNS. (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

author name string

Andre Michael Mueller

1

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

Bo Hyung Yoon

2

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

Saud Ahmed Sadiq

3

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Europe PubMed Central

PubMed publication ID

30 July 2017

language of work or name

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publication date

27 June 2014

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Europe PubMed Central

PubMed publication ID

30 July 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

30 July 2017

289

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Europe PubMed Central

PubMed publication ID

30 July 2017

33

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

22888-22899

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4132791/fullTextXML

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Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination

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Hyaluronan as an immune regulator in human diseases.

1 reference

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Th1 cytokines promote T-cell binding to antigen-presenting cells via enhanced hyaluronan production and accumulation at the immune synapse

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Hyaluronan synthesis is required for IL-2-mediated T cell proliferation

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Hyaluronan binding identifies the most proliferative activated and memory T cells.

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Hyaluronan anchored to activated CD44 on central nervous system vascular endothelial cells promotes lymphocyte extravasation in experimental autoimmune encephalomyelitis

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CXCL12 limits inflammation by localizing mononuclear infiltrates to the perivascular space during experimental autoimmune encephalomyelitis

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1 reference

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IL-17 is produced by some proinflammatory Th1/Th0 cells but not by Th2 cells

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CXCL12 expression within the CNS contributes to the resistance against experimental autoimmune encephalomyelitis in Albino Oxford rats

1 reference

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Identifiers

10.1074/JBC.M114.559583

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 July 2017

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