Oxidative stress by monoamine oxidases is causally involved in myofiber damage in muscular dystrophy (Q42934854)

11 November 2017

title

Oxidative stress by monoamine oxidases is causally involved in myofiber damage in muscular dystrophy (English)

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11 November 2017

Bert Blaauw

2

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Paola Braghetta

object named as

Paola Braghetta

5

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Luana Toniolo

object named as

Luana Toniolo

4

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Fabio Di Lisa

object named as

Fabio Di Lisa

8

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author name string

Sara Menazza

1

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11 November 2017

Tania Tiepolo

3

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11 November 2017

Barbara Spolaore

6

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11 November 2017

Carlo Reggiani

7

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11 November 2017

Paolo Bonaldo

9

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11 November 2017

Marcella Canton

10

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11 November 2017

publication date

17 August 2010

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11 November 2017

published in

Human Molecular Genetics

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11 November 2017

volume

19

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11 November 2017

issue

21

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11 November 2017

page(s)

4207-4215

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Muscular dystrophies involving the dystrophin-glycoprotein complex: an overview of current mouse models.

11 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Collagen VI deficiency induces early onset myopathy in the mouse: an animal model for Bethlem myopathy.

21 January 2018

1 reference

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Oxidative stress and the pathogenesis of muscular dystrophies

21 January 2018

1 reference

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Muscle damage in mdx (dystrophic) mice: role of calcium and reactive oxygen species.

21 January 2018

1 reference

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The role of free radicals in the pathophysiology of muscular dystrophy

21 January 2018

1 reference

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Flavocoxid counteracts muscle necrosis and improves functional properties in mdx mice: a comparison study with methylprednisolone.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

N-Acetylcysteine ameliorates skeletal muscle pathophysiology in mdx mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Genetic and pharmacologic inhibition of mitochondrial-dependent necrosis attenuates muscular dystrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Genetic ablation of cyclophilin D rescues mitochondrial defects and prevents muscle apoptosis in collagen VI myopathic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Mitochondrial dysfunction and apoptosis in myopathic mice with collagen VI deficiency.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

The therapeutic potential of monoamine oxidase inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Oxidative stress by monoamine oxidase mediates receptor-independent cardiomyocyte apoptosis by serotonin and postischemic myocardial injury

21 January 2018

1 reference

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Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Evidence of myofibrillar protein oxidation induced by postischemic reperfusion in isolated rat hearts

21 January 2018

1 reference

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Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization

21 January 2018

1 reference

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Akt activation prevents the force drop induced by eccentric contractions in dystrophin-deficient skeletal muscle

21 January 2018

1 reference

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Cell Therapy of -Sarcoglycan Null Dystrophic Mice Through Intra-Arterial Delivery of Mesoangioblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Dystrophin mutations predict cellular susceptibility to oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Cyclosporin A corrects mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathies

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Protein oxidation in aging, disease, and oxidative stress

21 January 2018

1 reference

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Physiological consequences of tropomyosin mutations associated with cardiac and skeletal myopathies

21 January 2018

1 reference

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A Mutant Tropomyosin That Causes Hypertrophic Cardiomyopathy Is Expressed In Vivo and Associated With an Increased Calcium Sensitivity

21 January 2018

1 reference

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Mitochondrial dysfunction in the pathogenesis of Ullrich congenital muscular dystrophy and prospective therapy with cyclosporins

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

The cyclophilin inhibitor Debio 025 normalizes mitochondrial function, muscle apoptosis and ultrastructural defects in Col6a1-/- myopathic mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Investigation of Debio 025, a cyclophilin inhibitor, in the dystrophic mdx mouse, a model for Duchenne muscular dystrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

The role of reactive oxygen species in integrin and matrix metalloproteinase expression and function

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Integrins engage mitochondrial function for signal transduction by a mechanism dependent on Rho GTPases

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Integrin-mediated cell adhesion and spreading engage different sources of reactive oxygen species.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

A CaPful of mechanisms regulating the mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Mitochondrial pathways for ROS formation and myocardial injury: the relevance of p66(Shc) and monoamine oxidase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Green tea extract and its major polyphenol (-)-epigallocatechin gallate improve muscle function in a mouse model for Duchenne muscular dystrophy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

Eccentric contractions lead to myofibrillar dysfunction in muscular dystrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDQ339

21 January 2018

Identifiers

DOI

10.1093/HMG/DDQ339

1 reference

11 November 2017

1 reference