NFATc1 expression in the developing heart valves is responsive to the RANKL pathway and is required for endocardial expression of cathepsin K. (Q52018491)

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English	NFATc1 expression in the developing heart valves is responsive to the RANKL pathway and is required for endocardial expression of cathepsin K.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

NFATc1 expression in the developing heart valves is responsive to the RANKL pathway and is required for endocardial expression of cathepsin K. (English)

1 reference

Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

Katherine E. Yutzey

2

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

author name string

Alexander W Lange

1

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

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publication date

1 April 2006

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

Developmental Biology

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

292

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

2

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

407-417

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Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

Spatial and temporal expression of the 72-kDa type IV collagenase (MMP-2) correlates with development and differentiation of valves in the embryonic avian heart.

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osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

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A field of myocardial-endocardial NFAT signaling underlies heart valve morphogenesis

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Regulation of vascular calcification by osteoclast regulatory factors RANKL and osteoprotegerin

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The collagenolytic activity of cathepsin K is unique among mammalian proteinases

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Cathepsin K: isolation and characterization of the murine cDNA and genomic sequence, the homologue of the human pycnodysostosis gene

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Cathepsin K Knockout Mice Develop Osteopetrosis Due to a Deficit in Matrix Degradation but Not Demineralization

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The incidence of congenital heart disease

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Transcriptional regulation by calcium, calcineurin, and NFAT

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Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand

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Atrioventricular valves of the mouse: II. Light and transmission electron microscopy.

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Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation

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Large scale gene expression analysis of osteoclastogenesis in vitro and elucidation of NFAT2 as a key regulator

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Defective valvulogenesis in HB-EGF and TACE-null mice is associated with aberrant BMP signaling

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A nonsense mutation in the cathepsin K gene observed in a family with pycnodysostosis

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NFATc1 mediates vascular endothelial growth factor-induced proliferation of human pulmonary valve endothelial cells

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Receptor activator of nuclear factor kappaB ligand and osteoprotegerin regulate aortic valve calcification

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Influence of receptor activator of nuclear factor kappa B on human aortic valve myofibroblasts.

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Acidification of the osteoclastic resorption compartment provides insight into the coupling of bone formation to bone resorption.

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Regulation of the calmodulin-stimulated protein phosphatase, calcineurin

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NFAT and Osterix cooperatively regulate bone formation

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Convergent signaling by acidosis and receptor activator of NF-kappaB ligand (RANKL) on the calcium/calcineurin/NFAT pathway in osteoclasts

1 reference

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OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

1 reference

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Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

1 reference

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DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice

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Nuclear factor of activated T-cells (NFAT) rescues osteoclastogenesis in precursors lacking c-Fos

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RANK is the essential signaling receptor for osteoclast differentiation factor in osteoclastogenesis

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Cell biology of cardiac cushion development.

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The transcription factor NF-ATc is essential for cardiac valve formation

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Form and function of developing heart valves: coordination by extracellular matrix and growth factor signaling

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Degradation of type IV collagen by matrix metalloproteinases is an important step in the epithelial-mesenchymal transformation of the endocardial cushions

1 reference

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Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts

1 reference

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The molecular triad OPG/RANK/RANKL: involvement in the orchestration of pathophysiological bone remodeling

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Biology of the TRANCE axis

1 reference

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Differential regulation of the expression of CD95 ligand, receptor activator of nuclear factor-kappa B ligand (RANKL), TNF-related apoptosis-inducing ligand (TRAIL), and TNF-alpha during T cell activation

1 reference

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Developmental anatomy of the heart: a tale of mice and man.

1 reference

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Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

1 reference

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7 January 2021

RANK-L induces the expression of NFATc1, but not of NFkappaB subunits during osteoclast formation

1 reference

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7 January 2021

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10.1016/J.YDBIO.2006.01.017

1 reference

Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 April 2018

http://europepmc.org/abstract/MED/16680826

ResearchGate publication ID

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