Rac1 and RhoA GTPases have antagonistic functions during N-cadherin-dependent cell-cell contact formation in C2C12 myoblasts (Q57370872)

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Marie Causeret

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Julien Cau

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Nicolas Taulet

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Sophie Charrasse

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Cécile Gauthier-Rouvière

Mechanisms of epithelial cell-cell adhesion and cell compaction revealed by high-resolution tracking of E-cadherin-green fluorescent protein

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publication date

September 2007

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number of pages

15

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based on heuristic

inferred from page(s)

published in

Biology of the Cell

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volume

99

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issue

9

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page(s)

503-517

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cites work

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Cell-cell adhesion and signalling

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The Small GTPases Rho and Rac Are Required for the Establishment of Cadherin-dependent Cell–Cell Contacts

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Regulation of cadherin function by Rho and Rac: modulation by junction maturation and cellular context.

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N-cadherin association with lipid rafts regulates its dynamic assembly at cell-cell junctions in C2C12 myoblasts

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N-cadherin-dependent cell-cell contact regulates Rho GTPases and beta-catenin localization in mouse C2C12 myoblasts

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Variation in cadherins and catenins expression is linked to both proliferation and transformation of Rhabdomyosarcoma

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RhoA GTPase regulates M-cadherin activity and myoblast fusion

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Force measurements in E-cadherin-mediated cell doublets reveal rapid adhesion strengthened by actin cytoskeleton remodeling through Rac and Cdc42.

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Differential effects of N-cadherin-mediated adhesion on the development of myotomal waves

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Adhesion molecules during somitogenesis in the avian embryo.

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Spatio-temporal regulation of Rac1 localization and lamellipodia dynamics during epithelial cell-cell adhesion

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Rho-family GTPases in cadherin-mediated cell-cell adhesion

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RhoG GTPase controls a pathway that independently activates Rac1 and Cdc42Hs

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N-cadherin promotes the commitment and differentiation of skeletal muscle precursor cells.

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Direct involvement of N-cadherin-mediated signaling in muscle differentiation

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Effects of regulated expression of mutant RhoA and Rac1 small GTPases on the development of epithelial (MDCK) cell polarity

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Regulation of myosin phosphatase by Rho and Rho-associated kinase (Rho-kinase)

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Cell adhesion molecules in myogenesis

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Recycling of E-cadherin: a potential mechanism for regulating cadherin dynamics

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Critical activities of Rac1 and Cdc42Hs in skeletal myogenesis: antagonistic effects of JNK and p38 pathways

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Participation of small GTPases Rac1 and Cdc42Hs in myoblast transformation

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Rhabdomyosarcoma--working out the pathways

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Cadherins promote skeletal muscle differentiation in three-dimensional cultures

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Rho signalling at a glance

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Myosin 2 is a key Rho kinase target necessary for the local concentration of E-cadherin at cell-cell contacts

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Rho-stimulated contractility contributes to the fibroblastic phenotype of Ras-transformed epithelial cells

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