Over-expression of Tfam improves the mitochondrial disease phenotypes in a mouse model system (Q84979590)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20816751%20AND%20SRC:MED&resulttype=core&format=json

13 February 2020

title

Over-expression of Tfam improves the mitochondrial disease phenotypes in a mouse model system (English)

1 reference

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13 February 2020

main subject

mitochondrial disease

1 reference

Satoshi Nishiyama

1

1 reference

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13 February 2020

Hiroshi Shitara

2

1 reference

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13 February 2020

Kazuto Nakada

3

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13 February 2020

Tomio Ono

4

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13 February 2020

Akitsugu Sato

5

1 reference

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13 February 2020

Hidenori Suzuki

6

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13 February 2020

Tetsuhiro Ogawa

7

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13 February 2020

Haruhiko Masaki

8

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13 February 2020

Jun-Ichi Hayashi

9

1 reference

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13 February 2020

Hiromichi Yonekawa

10

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13 February 2020

language of work or name

English

1 reference

National Center for Biotechnology Information

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=20816751

6 July 2024

publication date

15 September 2010

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20816751%20AND%20SRC:MED&resulttype=core&format=json

full work available at URL

13 February 2020

https://tsukuba.repo.nii.ac.jp/record/20610/files/BBRC_401-1.pdf

file format

Portable Document Format

online access status

open access

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A20816751

https://api.elsevier.com/content/article/PII:S0006291X10016645?httpAccept=text/xml

6 July 2024

1 reference

https://api.crossref.org/works/10.1016/J.BBRC.2010.08.143

https://api.elsevier.com/content/article/PII:S0006291X10016645?httpAccept=text/plain

6 July 2024

1 reference

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Biochemical and Biophysical Research Communications

6 July 2024

published in

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20816751%20AND%20SRC:MED&resulttype=core&format=json

13 February 2020

volume

401

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13 February 2020

issue

1

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13 February 2020

page(s)

26-31

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20816751%20AND%20SRC:MED&resulttype=core&format=json

Mitochondrial diseases in man and mouse

13 February 2020

cites work

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In vivo interaction between mitochondria carrying mtDNAs from different mouse species

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A mouse model of mitochondrial disease reveals germline selection against severe mtDNA mutations

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Generation of mice with mitochondrial dysfunction by introducing mouse mtDNA carrying a deletion into zygotes

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Rapid directional shift of mitochondrial DNA heteroplasmy in animal tissues by a mitochondrially targeted restriction endonuclease

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Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction

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Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA.

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Mitochondrial transcription factor A is necessary for mtDNA maintenance and embryogenesis in mice

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Maintenance of mitochondrial DNA copy number and expression are essential for preservation of mitochondrial function and cell growth

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Overexpression of mitochondrial transcription factor a ameliorates mitochondrial deficiencies and cardiac failure after myocardial infarction

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Mitochondrial transcription factor A regulates mtDNA copy number in mammals

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Molecular control of mitochondrial function in preimplantation mouse embryos.

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Gene therapy for progeny of mito-mice carrying pathogenic mtDNA by nuclear transplantation

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6 July 2024

Identifiers

1 reference

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13 February 2020

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