Oxidative stress pathways in the potentiation of noise-induced hearing loss by acrylonitrile (Q23910368)

2

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Alisa Nelson-Miller

3

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Sherry Fulton

4

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Laurence Fechter

University of Oklahoma Health Sciences Center

5

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language of work or name

1 reference

February 2007

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published in

Hearing Research

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volume

224

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issue

1-2

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page(s)

61-74

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performing organization

object named as

University of Oklahoma, Health Sciences Center, Oklahoma Center for Toxicology, Oklahoma City, Oklahoma

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NIOSHTIC-2

on focus list of Wikimedia project

2 February 2024

Wikimedia–NIOSH collaboration

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1 reference

25 September 2023

Assessment of cochlear function in mice: distortion-product otoacoustic emissions

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Species differences in acrylonitrile metabolism and toxicity between experimental animals and humans based on observations in human accidental poisonings

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Targeted mutation of the gene for cellular glutathione peroxidase (Gpx1) increases noise-induced hearing loss in mice

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Acrylonitrile potentiates noise-induced hearing loss in rat.

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Superoxide dismutase activity in the cochlea

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Frequency representation in the rat cochlea

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Dose and time-dependent protection of the antioxidant N-L-acetylcysteine against impulse noise trauma

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Distortion product otoacoustic emissions show exceptional resistance to noise exposure in MOLF/Ei mice.

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Acrylonitrile potentiates hearing loss and cochlear damage induced by moderate noise exposure in rats

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Protection from noise-induced lipid peroxidation and hair cell loss in the cochlea

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Role of glutathione in protection against noise-induced hearing loss

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Glutathione limits noise-induced hearing loss.

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Reduction of noise-induced hearing loss using L-NAC and salicylate in the chinchilla.

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Differential vulnerability of basal and apical hair cells is based on intrinsic susceptibility to free radicals

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Direct effects of intraperilymphatic reactive oxygen species generation on cochlear function

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Aminoglycosides activate oxygen metabolites production in the cochlea of mature and developing rats

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Disruption of cochlear potentials by chemical asphyxiants. Cyanide and carbon monoxide.

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Inhibition of microsomal oxidation of ethanol by pyrazole and 4-methylpyrazole in vitro. Increased effectiveness after induction by pyrazole and 4-methylpyrazole

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Prevention of impulse noise-induced hearing loss with antioxidants

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Acrylonitrile produces transient cochlear function loss and potentiates permanent noise-induced hearing loss

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The antidotal action of thiosulfate following acute nitroprusside infusion in dogs

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Double-Blind, Crossover, Placebo-Controlled Clinical Trial with L-Acetylcarnitine in Patients with Degenerative Cerebellar Ataxia

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Candidate's thesis: enhancing intrinsic cochlear stress defenses to reduce noise-induced hearing loss

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The role of antioxidants in protection from impulse noise

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Early elevation of cochlear reactive oxygen species following noise exposure

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Targeted deletion of the cytosolic Cu/Zn-superoxide dismutase gene (Sod1) increases susceptibility to noise-induced hearing loss

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The protective effects of allopurinol and superoxide dismutase on noise-induced cochlear damage.

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