Enhanced nitric oxide and reactive oxygen species production and damage after inhalation of silica (Q23911529)

HELD

1 reference

NIOSHTIC-2

NIOSH Division of Applied Research and Technology

25 September 2023

DART

1 reference

25 September 2023

2 references

19 July 2017

29 March 2020

2 references

19 July 2017

29 March 2020

1 reference

29 March 2020

1 reference

29 March 2020

1 reference

29 March 2020

1 reference

29 March 2020

1 reference

29 March 2020

reactive oxygen species

1 reference

12

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Dale W. Porter

1

Dale W. Porter

0 references

Ann F. Hubbs

4

Ann Hubbs

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Douglas Landsittel

10

Douglas Landsittel

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author name string

Lyndell Millecchia

2

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Victor A. Robinson

3

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Patsy Willard

5

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Donna Pack

6

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Dawn Ramsey

7

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Jeff McLaurin

8

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Amir Khan

9

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Alexander Teass

American Journal of Physiology - Lung Cellular and Molecular Physiology

11

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language of work or name

English

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publication date

15 March 2002

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published in

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volume

283

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issue

2

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page(s)

L485-L493

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on focus list of Wikimedia project

Wikimedia–NIOSH collaboration

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cites work

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Co-stimulatory effect of nitric oxide on endothelial NF-kappaB implies a physiological self-amplifying mechanism

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Freshly fractured quartz inhalation leads to enhanced lung injury and inflammation. Potential role of free radicals

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Oxidative stress in silicosis: evidence for the enhanced clearance of free radicals from whole lungs

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Nitric oxide modulates interleukin-1beta and tumour necrosis factor-alpha synthesis, and disease regression by alveolar macrophages in pulmonary tuberculosis.

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Effect of circulating neutrophil depletion on lung injury induced by inhaled silica particles

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A comparison of the inflammatory response of the lung to inhaled versus instilled particles in F344 rats

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Nitric oxide blocks nuclear factor-kappaB activation in alveolar macrophages

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Biological and statistical approaches to predicting human lung cancer risk from silica

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