MEN2A-RET-induced cellular transformation by activation of STAT3 (Q24291643)

1

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K Wojtachnio

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W Hagens

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E Vellenga

4

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C H Buys

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W Kruijer

Grb2 binding to the different isoforms of Ret tyrosine kinase.

7

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language of work or name

English

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publication date

30 August 2001

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published in

Oncogene

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volume

20

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issue

38

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page(s)

5350-8

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cites work

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Stat3 activation is required for cellular transformation by v-src

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Activation and association of Stat3 with Src in v-Src-transformed cell lines

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Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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Mitogenic signaling by Ret/ptc2 requires association with enigma via a LIM domain

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STAT3 orchestrates contradictory signals in cytokine-induced G1 to S cell-cycle transition

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RET gene and its implications for cancer

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STATs: signal transducers and activators of transcription

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Identification and functional characterization of a Smad binding element (SBE) in the JunB promoter that acts as a transforming growth factor-beta, activin, and bone morphogenetic protein-inducible enhancer.

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RET alternate splicing influences the interaction of activated RET with the SH2 and PTB domains of Shc, and the SH2 domain of Grb2

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ret transforming gene encodes a fusion protein homologous to tyrosine kinases

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Stat3 activation by Src induces specific gene regulation and is required for cell transformation

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Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation

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Stat3 plays an important role in oncogenic Ros- and insulin-like growth factor I receptor-induced anchorage-independent growth

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