The chemoattractant receptor-like protein C5L2 binds the C3a des-Arg77/acylation-stimulating protein (Q24293053)

4

object named as

Allan D Sniderman

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author name string

David Kalant

1

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Stuart A Cain

2

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Magdalena Maslowska

3

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Peter N Monk

Journal of Biological Chemistry

6

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language of work or name

English

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publication date

28 March 2003

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published in

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volume

278

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page(s)

11123-9

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issue

13

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cites work

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Inflammatory mediators in acute pancreatitis

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The human chemoattractant complement C5a receptor inhibits cyclic AMP accumulation through Gi and Gz proteins

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Acylation stimulating protein (ASP), an adipocyte autocrine: new directions

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Selective coupling of the human anaphylatoxin C5a receptor and alpha 16 in human kidney 293 cells

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Psychopharmacological activity of anaphylatoxin C3a in rat hypothalamus.

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Multiple signalling pathways in the C5a-induced expression of adhesion receptor Mac-1.

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Binding specificity and presynaptic action of anaphylatoxin C5a in rat brain

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Modulation of ligand selectivity by mutation of the first extracellular loop of the human C5a receptor

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A putative chemoattractant receptor, C5L2, is expressed in granulocyte and immature dendritic cells, but not in mature dendritic cells

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C3a receptor on dibutyryl-cAMP-differentiated U937 cells and human neutrophils: the human C3a receptor characterized by functional responses and 125I-C3a binding

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Mapping the ligand-binding site on the C5a receptor: arginine74 of C5a contacts aspartate282 of the C5a receptor

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The regulation of actin polymerization in differentiating U937 cells correlates with increased membrane levels of the pertussis-toxin-sensitive G-protein Gi2.

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Inhibition of in vitro natural killer activity by the third component of complement: role for the C3a fragment

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Mutation of glutamate 199 of the human C5a receptor defines a binding site for ligand distinct from the receptor N terminus

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The orphan receptor C5L2 has high affinity binding sites for complement fragments C5a and C5a des-Arg(74)

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A novel role of complement: mice deficient in the fifth component of complement (C5) exhibit impaired liver regeneration.

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