VLDL-induced triglyceride accumulation in human macrophages is mediated by modulation of LPL lipolytic activity in the absence of change in LPL mass (Q24294323)

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English	VLDL-induced triglyceride accumulation in human macrophages is mediated by modulation of LPL lipolytic activity in the absence of change in LPL mass	scientific article

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scholarly article

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VLDL-induced triglyceride accumulation in human macrophages is mediated by modulation of LPL lipolytic activity in the absence of change in LPL mass (English)

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lipoprotein lipase

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GOA release 2020-03-11

positive regulation of sequestering of triglyceride

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GOA release 2020-03-11

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based on heuristic

inferred from title

author name string

Dragana Milosavljevic

1

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Anatol Kontush

2

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Sabine Griglio

3

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Gilles Le Naour

4

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Joëlle Thillet

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M John Chapman

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language of work or name

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publication date

20 February 2003

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Biochimica et Biophysica Acta

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1631

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1

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51-60

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Selective retention of VLDL, IDL, and LDL in the arterial intima of genetically hyperlipidemic rabbits in vivo. Molecular size as a determinant of fractional loss from the intima-inner media

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Triglyceride-rich lipoproteins isolated by selected-affinity anti-apolipoprotein B immunosorption from human atherosclerotic plaque.

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Oxidized VLDL induces less triglyceride accumulation in J774 macrophages than native VLDL due to an impaired extracellular lipolysis

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Lipoprotein heterogeneity and apolipoprotein B metabolism

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Heterogeneity of apolipoprotein B-100-containing lipoproteins: what we have learnt from kinetic studies.

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Action of atorvastatin in combined hyperlipidemia : preferential reduction of cholesteryl ester transfer from HDL to VLDL1 particles

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Cholesterol accumulation in J774 macrophages induced by triglyceride-rich lipoproteins. Comparison of very low density lipoprotein from subjects with type III, IV, and V hyperlipoproteinemias.

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Regulation of lipoprotein lipase secretion in murine macrophages during foam cell formation in vitro. Effect of triglyceride-rich lipoproteins

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10.1016/S1388-1981(02)00355-4

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