Inhibition of the canonical Wnt pathway by Dickkopf-1 contributes to the neurodegeneration in 6-OHDA-lesioned rats (Q24296942)

negative regulation of canonical Wnt signaling pathway

GOA release 2020-03-11

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GOA release 2020-03-11

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Yaoyan Dun

1

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Gang Li

2

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Yang Yang

3

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Zhengguo Xiong

4

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Mei Feng

5

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Min Wang

6

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Yuan Zhang

7

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Jizhou Xiang

8

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Rong Ma

Activation of p53 signaling initiates apoptotic death in a cellular model of Parkinson's disease

9

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language of work or name

English

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publication date

13 September 2012

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published in

Neuroscience Letters

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volume

525

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issue

2

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page(s)

83-8

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cites work

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Lithium fails to protect dopaminergic neurons in the 6-OHDA model of Parkinson's disease.

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p53 in neuronal apoptosis

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Increased Dickkopf-1 expression accelerates bone cell apoptosis in femoral head osteonecrosis

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Behavioral and morphological effects of minocycline in the 6-hydroxydopamine rat model of Parkinson's disease

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Gene expression profiling in progressively MPTP-lesioned macaques reveals molecular pathways associated with sporadic Parkinson's disease

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Interferon-gamma regulates intestinal epithelial homeostasis through converging beta-catenin signaling pathways

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Histological, behavioural and neurochemical evaluation of medial forebrain bundle and striatal 6-OHDA lesions as rat models of Parkinson's disease

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Inflammation induction of Dickkopf-1 mediates chondrocyte apoptosis in osteoarthritic joint.

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Inhibition of Wnt signaling, modulation of Tau phosphorylation and induction of neuronal cell death by DKK1.

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Effects of gender on nigral gene expression and parkinson disease

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Reactive astrocytes and Wnt/β-catenin signaling link nigrostriatal injury to repair in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease

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Erythropoietin protects PC12 cells from beta-amyloid(25-35)-induced apoptosis via PI3K/Akt signaling pathway

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Signaling pathways mediating the neuroprotective effects of sex steroids and SERMs in Parkinson's disease

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Wnt signalling: antagonistic Dickkopfs

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Neuroinflammation in Parkinson's disease: a target for neuroprotection?

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Beta-catenin regulates expression of cyclin D1 in colon carcinoma cells

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Induction of the Wnt antagonist, Dickkopf-1, contributes to the development of neuronal death in models of brain focal ischemia.

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Kremen proteins are Dickkopf receptors that regulate Wnt/beta-catenin signalling

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Neuronal life and death: an essential role for the p53 family

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The role of p53 in neuronal cell death

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Dickkopf-1, an inhibitor of the Wnt signaling pathway, is induced by p53.

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Human Dkk-1, a gene encoding a Wnt antagonist, responds to DNA damage and its overexpression sensitizes brain tumor cells to apoptosis following alkylation damage of DNA

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The Wnt antagonist Dkk1 regulates intestinal epithelial homeostasis and wound repair.

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Target genes of beta-catenin-T cell-factor/lymphoid-enhancer-factor signaling in human colorectal carcinomas

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Akt participation in the Wnt signaling pathway through Dishevelled

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The Wnt antagonist Dickkopf-1 is regulated by Bmp signaling and c-Jun and modulates programmed cell death

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Reversal of thiamine deficiency-induced neurodegeneration

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Glycogen synthase kinase 3beta (GSK3beta) mediates 6-hydroxydopamine-induced neuronal death

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Alpha-Synuclein contributes to GSK-3beta-catalyzed Tau phosphorylation in Parkinson's disease models

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Induction of the Wnt inhibitor, Dickkopf-1, is associated with neurodegeneration related to temporal lobe epilepsy

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Dickkopf-1 Enhances Inflammatory Interaction Between Platelets and Endothelial Cells and Shows Increased Expression in Atherosclerosis

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Induction of Dickkopf-1, a negative modulator of the Wnt pathway, is associated with neuronal degeneration in Alzheimer's brain

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Role of Dickkopf-1, an antagonist of the Wnt/beta-catenin signaling pathway, in estrogen-induced neuroprotection and attenuation of tau phosphorylation

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Induction of Dickkopf-1, a negative modulator of the Wnt pathway, is required for the development of ischemic neuronal death.

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Circulating Dickkopf-1 is correlated with bone erosion and inflammation in rheumatoid arthritis

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

22902902

1 reference

Consolidated OpenCitations Corpus – April 2017