GADD34 induces p53 phosphorylation and p21/WAF1 transcription (Q24300442)

GOA release 2020-03-11

9

Ken-Ichi Isobe

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author name string

Ayako Yagi

1

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Yoshinori Hasegawa

2

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Hengyi Xiao

3

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Masataka Haneda

4

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Eiji Kojima

5

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Akihiko Nishikimi

6

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Tadao Hasegawa

7

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Kaoru Shimokata

Journal of Cellular Biochemistry

8

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language of work or name

English

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publication date

15 December 2003

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published in

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volume

90

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issue

6

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page(s)

1242-9

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cites work

Activation of Gadd34 by diverse apoptotic signals and suppression of its growth inhibitory effects by apoptotic inhibitors

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Sodium butyrate induces NIH3T3 cells to senescence-like state and enhances promoter activity of p21WAF/CIP1 in p53-independent manner

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Cloning of a GADD34-like gene that interacts with the zinc-finger transcription factor which binds to the p21(WAF) promoter

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Interaction between GADD34 and kinesin superfamily, KIF3A

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WAF1, a potential mediator of p53 tumor suppression

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Evidence for the interaction between Translin and GADD34 in mammalian cells

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Heterozygosity with respect to Zfp148 causes complete loss of fetal germ cells during mouse embryogenesis

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In vivo evidence for binding of p53 to consensus binding sites in the p21 and GADD45 genes in response to ionizing radiation

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Melanoma differentiation associated gene-7, mda-7/IL-24, selectively induces growth suppression, apoptosis and radiosensitization in malignant gliomas in a p53-independent manner

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Interaction between growth arrest-DNA damage protein 34 and Src kinase Lyn negatively regulates genotoxic apoptosis

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Mammalian GADD34, an apoptosis- and DNA damage-inducible gene

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The gamma134.5 protein of herpes simplex virus 1 has the structural and functional attributes of a protein phosphatase 1 regulatory subunit and is present in a high molecular weight complex with the enzyme in infected cells

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SWI/SNF complex interacts with tumor suppressor p53 and is necessary for the activation of p53-mediated transcription

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Feedback inhibition of the unfolded protein response by GADD34-mediated dephosphorylation of eIF2alpha

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Stress-induced gene expression requires programmed recovery from translational repression

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Repression of p53-mediated transcription by MDM2: a dual mechanism

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p53 alteration is a common event in the spontaneous immortalization of primary BALB/c murine embryo fibroblasts

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The gadd and MyD genes define a novel set of mammalian genes encoding acidic proteins that synergistically suppress cell growth

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Leukemic HRX fusion proteins inhibit GADD34-induced apoptosis and associate with the GADD34 and hSNF5/INI1 proteins

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ZBP-89 promotes growth arrest through stabilization of p53

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Growth arrest and DNA damage-inducible protein GADD34 assembles a novel signaling complex containing protein phosphatase 1 and inhibitor 1.

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Mammalian genes coordinately regulated by growth arrest signals and DNA-damaging agents

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