Dysregulation of HSG triggers vascular proliferative disorders (Q24301690)

negative regulation of Ras protein signal transduction

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

Kuang-Hueih Chen

1

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Xiaomei Guo

2

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Dalong Ma

3

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Yanhong Guo

4

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Qian Li

5

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Dongmei Yang

6

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Pengfei Li

7

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Xiaoyan Qiu

8

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Shaojun Wen

9

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Rui-Ping Xiao

10

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Jian Tang

11

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language of work or name

English

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publication date

September 2004

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published in

Nature Cell Biology

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volume

6

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issue

9

Epiregulin is a potent vascular smooth muscle cell-derived mitogen induced by angiotensin II, endothelin-1, and thrombin

5

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31 January 2017

872-83

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Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals

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Sirolimus-Eluting Stents versus Standard Stents in Patients with Stenosis in a Native Coronary Artery

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Mice lacking p27(Kip1) display increased body size, multiple organ hyperplasia, retinal dysplasia, and pituitary tumors

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Cardiovascular disease increasing in developing countries

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Cell cycle progression: new therapeutic target for vascular proliferative disease

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Recombinant platelet-derived growth factor B gene expression in porcine arteries induce intimal hyperplasia in vivo

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Recombinant fibroblast growth factor-1 promotes intimal hyperplasia and angiogenesis in arteries in vivo

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Cellular ras activity is required for passage through multiple points of the G0/G1 phase in BALB/c 3T3 cells

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The p42/p44 mitogen-activated protein kinase activation triggers p27Kip1 degradation independently of CDK2/cyclin E in NIH 3T3 cells.

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Myc and Ras collaborate in inducing accumulation of active cyclin E/Cdk2 and E2F.

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Ras links growth factor signaling to the cell cycle machinery via regulation of cyclin D1 and the Cdk inhibitor p27KIP1.

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Developmentally regulated mitochondrial fusion mediated by a conserved, novel, predicted GTPase.

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Development of a strain of spontaneously hypertensive rats

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Impaired ceramide signalling in spontaneously hypertensive rat vascular smooth muscle: a possible mechanism for augmented cell proliferation

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The spontaneously hypertensive rat as a model of the transition from compensated left ventricular hypertrophy to failure

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Significance of quiescent smooth muscle migration in the injured rat carotid artery

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Time course of smooth muscle cell proliferation in the intima and media of arteries following experimental angioplasty

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Germ-line transmission of a mutated p53 gene in a cancer-prone family with Li-Fraumeni syndrome

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Requirement of p27Kip1 for restriction point control of the fibroblast cell cycle

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A syndrome of multiorgan hyperplasia with features of gigantism, tumorigenesis, and female sterility in p27(Kip1)-deficient mice

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Rb function in cell-cycle regulation and apoptosis

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Cellular targets for activation by the E2F1 transcription factor include DNA synthesis- and G1/S-regulatory genes

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Recent progress in gene therapy for cardiovascular disease

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Cytostatic gene therapy for vascular proliferative disorders with a constitutively active form of the retinoblastoma gene product

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Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty

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Gene therapy for vascular smooth muscle cell proliferation after arterial injury

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Novel methods for adenovirus-mediated gene transfer to blood vessels in vivo

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Adenovirus-mediated heme oxygenase-1 gene delivery inhibits injury-induced vascular neointima formation

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Effect of percutaneous adenovirus-mediated Gax gene delivery to the arterial wall in double-injured atheromatous stented rabbit iliac arteries

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Transcriptional targeting of replication-defective adenovirus transgene expression to smooth muscle cells in vivo.

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The Third Intracellular Loop and the Carboxyl Terminus of β2-Adrenergic Receptor Confer Spontaneous Activity of the Receptor

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A modified alkaline lysis method for the preparation of highly purified plasmid DNA from Escherichia coli

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Identifiers

DOI

10.1038/NCB1161

1 reference

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

3789459

1015000233

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Fatcat ID

release_3elix6s2xze2vdygkjuep5b3rm

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Fatcat

https://api.fatcat.wiki/v0/release/3elix6s2xze2vdygkjuep5b3rm

24 November 2022

mapped directly with Wikidata item

3789459

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference