Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2 (Q24310216)

GOA release 2020-03-11

cell-cell signaling

1 reference

GOA release 2020-03-11

M Vikkula

1

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Douglas A Marchuk

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ORCID Public Data File 2021

author name string

L M Boon

2

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K L Carraway

3

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J T Calvert

4

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A J Diamonti

5

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B Goumnerov

6

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K A Pasyk

7

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D A Marchuk

8

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M L Warman

9

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L C Cantley

10

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J B Mulliken

11

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B R Olsen

Human venous endothelium can promote intimal hyperplasia in a paracrine manner

12

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language of work or name

English

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publication date

27 December 1996

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published in

Cell

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volume

87

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page(s)

1181-90

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issue

7

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cites work

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An activated form of transforming growth factor beta is produced by cocultures of endothelial cells and pericytes

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Assignment of a locus for dominantly inherited venous malformations to chromosome 9p

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Growth factor effects on cells of the vascular wall: a survey

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Isolation of angiopoietin-1, a ligand for the TIE2 receptor, by secretion-trap expression cloning

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Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts

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Assignment of the endothelial-specific protein receptor tyrosine kinase gene (TEK) to human chromosome 9p21

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Dominant-negative and targeted null mutations in the endothelial receptor tyrosine kinase, tek, reveal a critical role in vasculogenesis of the embryo

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Vascularization of the mouse embryo: a study of flk-1, tek, tie, and vascular endothelial growth factor expression during development.

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The effect of endothelial cell coculture on smooth muscle cell proliferation

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A gene for familial venous malformations maps to chromosome 9p in a second large kindred

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Attachment of smooth muscle cells to collagen and their migration toward platelet-derived growth factor

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Platelet-derived growth factor is a chemoattractant for vascular smooth muscle cells

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Mutations in the activin receptor-like kinase 1 gene in hereditary haemorrhagic telangiectasia type 2.

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Expression of the fms-like tyrosine kinase 4 gene becomes restricted to lymphatic endothelium during development.

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Neural crest and cardiovascular patterning.

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A two-hit model for developmental defects in Gorlin syndrome

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Mice deficient for PDGF B show renal, cardiovascular, and hematological abnormalities

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Endoglin, a TGF-beta binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1

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Structure of the FGF receptor tyrosine kinase domain reveals a novel autoinhibitory mechanism

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Electron microscopic study of the prenatal development of the thoracic aorta in the rat.

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Graded activation of fibroblast growth factor receptor 3 by mutations causing achondroplasia and thanatophoric dysplasia

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Embryonic origins and assembly of blood vessels

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Inhibition of capillary endothelial cell growth by pericytes and smooth muscle cells

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Matrix-specific effect of endothelial control of smooth muscle cell migration

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Endothelial cell modulation of smooth muscle cell morphology and organizational growth pattern

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Distinct roles of the receptor tyrosine kinases Tie-1 and Tie-2 in blood vessel formation

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A single ataxia telangiectasia gene with a product similar to PI-3 kinase

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Catalytic specificity of protein-tyrosine kinases is critical for selective signalling

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Cellular markers that distinguish the phases of hemangioma during infancy and childhood

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Profound ligand-independent kinase activation of fibroblast growth factor receptor 3 by the activation loop mutation responsible for a lethal skeletal dysplasia, thanatophoric dysplasia type II

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B-type receptor for platelet-derived growth factor mediates a chemotactic response by means of ligand-induced activation of the receptor protein-tyrosine kinase

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Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

1887076

1887076

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference