A mechanistic link between an inherited and an acquired cardiac arrhythmia: HERG encodes the IKr potassium channel (Q24316252)

positive regulation of potassium ion transmembrane transport

GOA release 2020-03-11

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GOA release 2020-03-11

potassium

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author name string

M C Sanguinetti

1

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C Jiang

2

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M E Curran

3

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M T Keating

Oogenesis in Xenopus laevis (Daudin). I. Stages of oocyte development in laboratory maintained animals

4

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language of work or name

English

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publication date

21 April 1995

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published in

Cell

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volume

81

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page(s)

299-307

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issue

2

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cites work

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A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome

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SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome

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Strong voltage-dependent inward rectification of inward rectifier K+ channels is caused by intracellular spermine.

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Determination of the subunit stoichiometry of a voltage-activated potassium channel

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Ether-à-go-go encodes a voltage-gated channel permeable to K+ and Ca2+ and modulated by cAMP.

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Cloning and functional expression of a rat heart KATP channel

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POTENTIAL, IMPEDANCE, AND RECTIFICATION IN MEMBRANES

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Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents

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Modulation of the delayed rectifier, IK, by cadmium in cat ventricular myocytes

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Isoproterenol antagonizes prolongation of refractory period by the class III antiarrhythmic agent E-4031 in guinea pig myocytes. Mechanism of action

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Rate-dependent prolongation of cardiac action potentials by a methanesulfonanilide class III antiarrhythmic agent. Specific block of rapidly activating delayed rectifier K+ current by dofetilide

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Modulation by cAMP of a slowly activating potassium channel expressed in Xenopus oocytes

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