The antiapoptotic protein AAC-11 interacts with and regulates Acinus-mediated DNA fragmentation (Q24318700)

GOA release 2020-03-11

author name string

Patricia Rigou

1

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Valeria Piddubnyak

2

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Audrey Faye

3

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Jean-Christophe Rain

4

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Laurence Michel

5

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Fabien Calvo

6

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Jean-Luc Poyet

Functional identification of Api5 as a suppressor of E2F-dependent apoptosis in vivo

7

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language of work or name

English

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publication date

3 June 2009

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published in

The EMBO Journal

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volume

28

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issue

11

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page(s)

1576-88

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cites work

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AAC-11, a novel cDNA that inhibits apoptosis after growth factor withdrawal

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Loss of Acinus inhibits oligonucleosomal DNA fragmentation but not chromatin condensation during apoptosis

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The antiapoptotic protein Api5 and its partner, high molecular weight FGF2, are up-regulated in B cell chronic lymphoid leukemia

21 January 2018

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FIF [Fibroblast Growth Factor-2 (FGF-2)-Interacting-Factor], a Nuclear Putatively Antiapoptotic Factor, Interacts Specifically with FGF-2

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https://api.crossref.org/works/10.1038%2FEMBOJ.2009.106

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29 November 2018

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Expression of the antiapoptosis gene, AAC-11, as a prognosis marker in non-small cell lung cancer

29 November 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/19387494

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Differential cytotoxic pathways of topoisomerase I and II anticancer agents after overexpression of the E2F-1/DP-1 transcription factor complex

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/19387494

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Phosphorus distribution in perichromatin granules and surrounding nucleoplasm as visualized by electron spectroscopic imaging

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