In vitro inhibition of transthyretin aggregate-induced cytotoxicity by full and peptide derived forms of the soluble receptor for advanced glycation end products (RAGE) (Q24337632)

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English	In vitro inhibition of transthyretin aggregate-induced cytotoxicity by full and peptide derived forms of the soluble receptor for advanced glycation end products (RAGE)	scientific article

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In vitro inhibition of transthyretin aggregate-induced cytotoxicity by full and peptide derived forms of the soluble receptor for advanced glycation end products (RAGE) (English)

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Filipe A Monteiro

object named as

Filipe Almeida Monteiro

1

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Mónica M. Sousa

object named as

Mónica Mendes Sousa

3

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object named as

Isabel Cardoso

2

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Maria João Saraiva

object named as

Maria João Saraiva

4

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language of work or name

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publication date

12 June 2006

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580

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14

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3451-6

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Familial amyloid polyneuropathy: an electron microscope study of the peripheral nerve in five cases. I. Interstitial changes

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Familial amyloid polyneuropathy: an electron microscope study of the peripheral nerve in five cases. II. Nerve fibre changes

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Transthyretin mutations in hyperthyroxinemia and amyloid diseases

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Amyloid fibril protein in familial amyloidotic polyneuropathy, Portuguese type. Definition of molecular abnormality in transthyretin (prealbumin).

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RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

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Advanced glycation end products (AGE) and their receptor (RAGE) in the brain of patients with Creutzfeldt-Jakob disease with prion plaques

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Receptor-dependent cell stress and amyloid accumulation in systemic amyloidosis

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RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

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N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

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RAGE is a multiligand receptor of the immunoglobulin superfamily: implications for homeostasis and chronic disease

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Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts

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Blockade of receptor for advanced glycation end-products restores effective wound healing in diabetic mice.

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N -Glycans on the receptor for advanced glycation end products influence amphoterin binding and neurite outgrowth

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Requirement for p38 and p44/p42 mitogen-activated protein kinases in RAGE-mediated nuclear factor-kappaB transcriptional activation and cytokine secretion

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Advanced glycation end product ligands for the receptor for advanced glycation end products: biochemical characterization and formation kinetics

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Human serum amyloid P component is an invariant constituent of amyloid deposits and has a uniquely homogeneous glycostructure

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Purification and characterization of mouse soluble receptor for advanced glycation end products (sRAGE)

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Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation

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Activation of ERK1/2 MAP kinases in familial amyloidotic polyneuropathy

1 reference

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Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases

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Thyroxine binding to transthyretin Met 119. Comparative studies of different heterozygotic carriers and structural analysis

1 reference

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based on heuristic

inferred from DOI database lookup

Strjcture of human plasma prealbumin at 2-5 A resolution. A preliminary report on the polypeptide chain conformation, quaternary structure and thyroxine binding

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based on heuristic

inferred from DOI database lookup

‘In vitro’ amyloid fibril formation from transthyretin: the influence of ions and the amyloidogenicity of TTR variants

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RAGE and amyloid beta interactions: atomic force microscopy and molecular modeling

1 reference

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based on heuristic

inferred from DOI database lookup

Deposition of transthyretin in early stages of familial amyloidotic polyneuropathy: evidence for toxicity of nonfibrillar aggregates

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.05.020

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based on heuristic

inferred from DOI database lookup

Interaction of the receptor for advanced glycation end products (RAGE) with transthyretin triggers nuclear transcription factor kB (NF-kB) activation

1 reference

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based on heuristic

inferred from DOI database lookup

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.05.020

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based on heuristic

inferred from DOI database lookup

The crystal structure of amyloidogenic Leu55 --> Pro transthyretin variant reveals a possible pathway for transthyretin polymerization into amyloid fibrils

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inferred from DOI database lookup

Antagonism of RAGE suppresses peripheral nerve regeneration

1 reference

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based on heuristic

inferred from DOI database lookup

The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses

1 reference

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based on heuristic

inferred from DOI database lookup

Receptor for advanced glycation end products (RAGE) regulates sepsis but not the adaptive immune response

1 reference

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Familial amyloid polyneuropathy: receptor for advanced glycation end products-dependent triggering of neuronal inflammatory and apoptotic pathways.

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

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10.1016/J.FEBSLET.2006.05.020

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