D-cycloserine deters reacquisition of cocaine self-administration by augmenting extinction learning (Q24626679)

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Jonathan J Szalay

2

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Cindy Achat-Mendes

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Donna M Platt

4

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Roger D Spealman

6

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Kathleen M Kantak

Effects of the partial glycine agonist D-cycloserine on cognitive functioning in chronic low dose MPTP-treated monkeys

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language of work or name

English

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publication date

January 2010

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published in

Neuropsychopharmacology

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volume

35

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issue

2

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page(s)

357-67

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cites work

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D-cycloserine accelerates the extinction of cocaine-induced conditioned place preference in C57bL/c mice

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Intra-amygdala and systemic antagonism of NMDA receptors prevents the reconsolidation of drug-associated memory and impairs subsequently both novel and previously acquired drug-seeking behaviors.

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D-cycloserine facilitates context-specific fear extinction learning

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NMDA receptor blockade in the basolateral amygdala disrupts consolidation of stimulus-reward memory and extinction learning during reinstatement of cocaine-seeking in an animal model of relapse

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Effects of D-cycloserine on extinction: translation from preclinical to clinical work

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Augmentation of exposure therapy with D-cycloserine for social anxiety disorder.

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Cognitive enhancers as adjuncts to psychotherapy: use of D-cycloserine in phobic individuals to facilitate extinction of fear

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Modulation of cocaine and food self-administration by low- and high-efficacy D1 agonists in squirrel monkeys

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D-cycloserine potentiates the reconsolidation of cocaine-associated memories

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D-cycloserine: a ligand for the N-methyl-D-aspartate coupled glycine receptor has partial agonist characteristics

30 May 2018

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Failure of d-cycloserine to reverse cognitive disruption induced by scopolamine or phencyclidine in primates

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d-Cycloserine facilitates extinction of a cocaine-induced conditioned place preference.

30 May 2018

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Decreased gray matter concentration in the insular, orbitofrontal, cingulate, and temporal cortices of cocaine patients

21 January 2018

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Potential role for the hypothalamo-pituitary-adrenal axis in the conditioned reinforcer-induced reinstatement of extinguished cocaine seeking in rats

21 January 2018

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Effects of brief and long maternal separations on the HPA axis activity and the performance of rats on context and tone fear conditioning.

21 January 2018

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Increasing the efficacy of cue exposure treatment in preventing relapse of addictive behavior.

21 January 2018

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Posttraining handling facilitates memory for auditory-cue fear conditioning in rats.

21 January 2018

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Influence of cocaine self-administration on learning related to prefrontal cortex or hippocampus functioning in rats

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D-cycloserine facilitates extinction the first time but not the second time: an examination of the role of NMDA across the course of repeated extinction sessions

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Effects of D-cycloserine on extinction of conditioned freezing

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D-cycloserine and the facilitation of extinction of conditioned fear: consequences for reinstatement

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D-cycloserine facilitates extinction of learned fear: effects on reacquisition and generalized extinction

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Decreased absolute amygdala volume in cocaine addicts

21 January 2018

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The facilitative effects of D-cycloserine on extinction of a cocaine-induced conditioned place preference can be long lasting and resistant to reinstatement

21 January 2018

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D-cycloserine facilitates extinction but does not eliminate renewal of the conditioned emotional response.

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Glutamate NMDA receptors within the amygdala participate in the modulatory effect of glucocorticoids on extinction of conditioned fear in rats.

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Efficacy of d-cycloserine for enhancing response to cognitive-behavior therapy for panic disorder

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Facilitation of conditioned fear extinction by systemic administration or intra-amygdala infusions of D-cycloserine as assessed with fear-potentiated startle in rats.

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D-cycloserine, a partial agonist at the glycine site coupled to N-methyl-D-aspartate receptors, improves visual recognition memory in rhesus monkeys.

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Combined Pharmacotherapy and Cognitive-Behavioral Therapy for Anxiety Disorders: Medication Effects, Glucocorticoids, and Attenuated Treatment Outcomes

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