Intestinal alkaline phosphatase is a gut mucosal defense factor maintained by enteral nutrition (Q24656115)

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William G Austen

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Xiaobo Zhang

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Gitonga Munene

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Golam Mostafa

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Shaluk Biswas

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Michael McCormack

7

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Kyle R Eberlin

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John T Nguyen

9

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Hamit S Tatlidede

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H Shaw Warren

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Sonoko Narisawa

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Jose L Millán

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Richard A Hodin

Proceedings of the National Academy of Sciences of the United States of America

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language of work or name

English

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publication date

4 March 2008

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published in

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volume

105

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issue

9

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page(s)

3551-6

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cites work

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Innate defenses of the intestinal epithelial barrier

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Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury

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Accelerated Fat Absorption in Intestinal Alkaline Phosphatase Knockout Mice

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Rat enterocytes secrete SLPs containing alkaline phosphatase and cubilin in response to corn oil feeding

28 September 2017

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Intestinal epithelial hyperpermeability: update on the pathogenesis of gut mucosal barrier dysfunction in critical illness

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Man the barrier! Strategic defences in the intestinal mucosa

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The gastrointestinal ecosystem: a precarious alliance among epithelium, immunity and microbiota

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Curcumin blocks cytokine-mediated NF-kappa B activation and proinflammatory gene expression by inhibiting inhibitory factor I-kappa B kinase activity

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Evolution of alkaline phosphatases in primates

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The role of the liver in clearance of glycoproteins from the general circulation, with special reference to intestinal alkaline phosphatase

28 September 2017

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The gut: a central organ after surgical stress

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Gut decontamination reduces bowel ischemia-induced lung injury in rats

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Temporal pattern of rat small intestinal gene expression with refeeding.

30 May 2018

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Synthesis and parallel secretion of rat intestinal alkaline phosphatase and a surfactant-like particle protein

30 May 2018

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A malachite green procedure for orthophosphate determination and its use in alkaline phosphatase-based enzyme immunoassay.

30 May 2018

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The gastrointestinal tract. The "undrained abscess" of multiple organ failure

30 May 2018

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Crossref

https://api.crossref.org/works/10.1073%2FPNAS.0712140105

Calf intestinal alkaline phosphatase, a novel therapeutic drug for lipopolysaccharide (LPS)-mediated diseases, attenuates LPS toxicity in mice and piglets.

21 January 2018

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Crossref

https://api.crossref.org/works/10.1073%2FPNAS.0712140105

Enterocyte response to ischemia is dependent on differentiation state

21 January 2018

1 reference

Crossref

https://api.crossref.org/works/10.1073%2FPNAS.0712140105

On the role and fate of LPS-dephosphorylating activity in the rat liver

21 January 2018

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2265168

Pattern of rat intestinal brush-border enzyme gene expression changes with epithelial growth state.

28 November 2018

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2265168

Intestinal mucosal lesion in low-flow states. I. A morphological, hemodynamic, and metabolic reappraisal

28 November 2018

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12 December 2020

Multiple-organ-failure syndrome

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12 December 2020

Benefits of immediate jejunostomy feeding after major abdominal trauma--a prospective, randomized study

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12 December 2020

Cellular growth state differentially regulates enterocyte gene expression in butyrate-treated HT-29 cells

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12 December 2020

Parenteral nutrition is associated with intestinal morphologic and functional changes in humans

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12 December 2020

Elimination of alkaline phosphatases from circulation by the galactose receptor. Different isoforms are cleared at various rates

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12 December 2020

Intestinal reperfusion injury is mediated by IgM and complement

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12 December 2020

The pro-inflammatory cytokines, IL-1beta and TNF-alpha, inhibit intestinal alkaline phosphatase gene expression

1 reference

12 December 2020

Identifiers

DOI

10.1073/PNAS.0712140105

1 reference

2367561

ADS bibcode

2008PNAS..105.3551G

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2367561

1 reference

1 reference

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