Effects of ciprofloxacin on eucaryotic pyrimidine nucleotide biosynthesis and cell growth (Q24677043)

2

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A B Pardee

3

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S F Schlossman

4

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T F Tedder

Antimicrobial Agents and Chemotherapy

5

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language of work or name

English

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publication date

May 1987

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published in

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volume

31

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issue

5

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page(s)

774-9

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cites work

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Targeted and Untargeted Mutagenesis by Various Inducers of SOS Functions in Escherichia coli

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Mitochondria contain a distinct DNA topoisomerase

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N-(phosphonacetyl)-L-aspartate, a potent transition state analog inhibitor of aspartate transcarbamylase, blocks proliferation of mammalian cells in culture.

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Novobiocin inhibition of DNA excision repair may occur through effects on mitochondrial structure and ATP metabolism, not on repair topoisomerases.

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Selective inhibition of pyrimidine synthesis and depletion of nucleotide pools by N-(phosphonacetyl)-L-aspartate

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The in-vitro activity of ciprofloxacin compared with that of norfloxacin and nalidixic acid.

30 May 2018

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Comparative in vitro activity of three new quinolone antibiotics against recent clinical isolates.

30 May 2018

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30 May 2018

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Banding in human chromosomes treated with trypsin

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Increase of UV-induced DNA repair synthesis during blast transformation of human lymphocytes

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Enhancement of 5-fluorouracil incorporation into human lymphoblast ribonucleic acid

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Inhibitor evidence for allosteric interaction in the replitase multienzyme complex

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Quinolone antibiotics inhibit eucaryotic DNA polymerase alpha and beta, terminal deoxynucleotidyl transferase but not DNA ligase

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DNA-protein cross-linking by ultraviolet radiation in normal human and xeroderma pigmentosum fibroblasts

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