NKB1: a natural killer cell receptor involved in the recognition of polymorphic HLA-B molecules (Q24679688)

1

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J Gumperz

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P Parham

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J H Phillips

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L L Lanier

Journal of Experimental Medicine

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language of work or name

English

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publication date

1 August 1994

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published in

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volume

180

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issue

2

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page(s)

537-43

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cites work

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Specificity of HLA class I antigen recognition by human NK clones: evidence for clonal heterogeneity, protection by self and non-self alleles, and influence of the target cell type

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HLA-C is the inhibitory ligand that determines dominant resistance to lysis by NK1- and NK2-specific natural killer cells

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Alloantigen recognition by two human natural killer cell clones is associated with HLA-C or a closely linked gene

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Involvement of HLA class I alleles in natural killer (NK) cell-specific functions: expression of HLA-Cw3 confers selective protection from lysis by alloreactive NK clones displaying a defined specificity (specificity 2)

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Interleukin 2 activation of natural killer cells rapidly induces the expression and phosphorylation of the Leu-23 activation antigen

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Demonstration by class I gene transfer that reduced susceptibility of human cells to natural killer cell-mediated lysis is inversely correlated with HLA class I antigen expression

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Identification of four subsets of human CD3-CD16+ natural killer (NK) cells by the expression of clonally distributed functional surface molecules: correlation between subset assignment of NK clones and ability to mediate specific alloantigen recogn

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The HLA-A,B "negative" mutant cell line C1R expresses a novel HLA-B35 allele, which also has a point mutation in the translation initiation codon

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Subpopulations of human natural killer cells defined by expression of the Leu-7 (HNK-1) and Leu-11 (NK-15) antigens

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PubMed

https://pubmed.ncbi.nlm.nih.gov/8046332

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Chromosomal location of the Ly-49 (A1, YE1/48) multigene family. Genetic association with the NK 1.1 antigen

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/8046332

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1084/JEM.180.2.537

1 reference

1 reference

1 reference

PubMed publication ID

8046332

1 reference