Learning from myocarditis: mimicry, chaos and black holes (Q26992048)

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2014

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Molecular mimicry, bystander activation, or viral persistence: infections and autoimmune disease.

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Nasal cardiac myosin peptide treatment and OX40 blockade protect mice from acute and chronic virally-induced myocarditis

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Gonadectomy of male BALB/c mice increases Tim-3(+) alternatively activated M2 macrophages, Tim-3(+) T cells, Th2 cells and Treg in the heart during acute coxsackievirus-induced myocarditis

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From myocarditis to cardiomyopathy: mechanisms of inflammation and cell death: learning from the past for the future

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Interleukin-17A Is Dispensable for Myocarditis but Essential for the Progression to Dilated Cardiomyopathy

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12 December 2020

Susceptibility to autoimmune myocarditis is associated with intrinsic differences in CD4(+) T cells

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Nasal administration of cardiac myosin suppresses autoimmune myocarditis in mice

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Myosin-induced acute myocarditis is a T cell-mediated disease

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Two autoimmune diabetes loci influencing T cell apoptosis control susceptibility to experimental autoimmune myocarditis

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Cardiac myosin and autoimmune myocarditis

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Rat dilated cardiomyopathy after autoimmune giant cell myocarditis

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