IL-22 Protects against Tissue Damage during Cutaneous Leishmaniasis (Q27301071)

1

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David Artis

6

object named as

David Artis

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author name string

Lucas Carvalho

3

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Edgar M Carvalho

4

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Elizabeth A Grice

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Phillip Scott

Creative Commons Attribution 4.0 International

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language of work or name

English

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publication date

2015

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published in

PLOS One

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volume

10

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issue

8

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page(s)

e0134698

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copyright license

start time

18 August 2015

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Successful therapy of visceral leishmaniasis with curdlan involves T-helper 17 cytokines

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Mucosal Leishmaniasis Patients Display an Activated Inflammatory T-cell Phenotype Associated with a Nonbalanced Monocyte Population

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Wound healing response is a major contributor to the severity of cutaneous leishmaniasis in the ear model of infection

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Disease control in cutaneous leishmaniasis is independent of IL-22

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Selection of a skin test antigen for American visceral leishmaniasis

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IL-1-induced inflammation promotes development of leishmaniasis in susceptible BALB/c mice

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Skin Inflammation Induced by the Synergistic Action of IL-17A, IL-22, Oncostatin M, IL-1 , and TNF- Recapitulates Some Features of Psoriasis

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The Effects of IL-20 Subfamily Cytokines on Reconstituted Human Epidermis Suggest Potential Roles in Cutaneous Innate Defense and Pathogenic Adaptive Immunity in Psoriasis

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IL-22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis

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