Nucleotide-oligomerization-domain-2 affects commensal gut microbiota composition and intracerebral immunopathology in acute Toxoplasma gondii induced murine ileitis (Q27314835)

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scientific article (publication date: 2014)

Language	Label	Description	Also known as
English	Nucleotide-oligomerization-domain-2 affects commensal gut microbiota composition and intracerebral immunopathology in acute Toxoplasma gondii induced murine ileitis	scientific article (publication date: 2014)

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scholarly article

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Nucleotide-oligomerization-domain-2 affects commensal gut microbiota composition and intracerebral immunopathology in acute Toxoplasma gondii induced murine ileitis (English)

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Toxoplasma gondii

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1 reference

based on heuristic

inferred from title

Stefan Bereswill

8

object named as

Stefan Bereswill

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Markus Heimesaat

1

object named as

Markus M Heimesaat

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author name string

Ildiko R Dunay

2

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Marie Alutis

3

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André Fischer

4

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Luisa Möhle

5

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Ulf B Göbel

6

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Anja A Kühl

7

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language of work or name

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publication date

2014

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9

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8

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e105120

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copyright license

Creative Commons Attribution 4.0 International

20 August 2014

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April 2022 Public Data File from Crossref

copyright status

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Novel murine infection models provide deep insights into the "ménage à trois" of Campylobacter jejuni, microbiota and host innate immunity

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NOD-like receptors (NLRs): bona fide intracellular microbial sensors

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Microbial influences in inflammatory bowel diseases

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Survey of extra-intestinal immune responses in asymptomatic long-term Campylobacter jejuni-infected mice

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Can microbiota transplantation abrogate murine colonization resistance against Campylobacter jejuni?

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Small intestinal nematode infection of mice is associated with increased enterobacterial loads alongside the intestinal tract

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The innate immune protein Nod2 binds directly to MDP, a bacterial cell wall fragment

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28 September 2017

Intrinsic expression of Nod2 in CD4+ T lymphocytes is not necessary for the development of cell-mediated immunity and host resistance to Toxoplasma gondii.

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Toxoplasma gondii rhoptry kinase ROP16 activates STAT3 and STAT6 resulting in cytokine inhibition and arginase-1-dependent growth control

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Nod2: a key regulator linking microbiota to intestinal mucosal immunity

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Commensal Bacteroides species induce colitis in host-genotype-specific fashion in a mouse model of inflammatory bowel disease

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28 September 2017

Immunology of Toxoplasma gondii

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Bifidobacterium animalis AHC7 protects against pathogen-induced NF-κB activation in vivo.

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Microbiota and probiotics: effects on human health

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IL-10 production by CD4+ effector T cells: a mechanism for self-regulation

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The induction of colitis and ileitis in mice is associated with marked increases in intestinal concentrations of stimulants of TLRs 2, 4, and 5

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Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.

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T cell-intrinsic role of Nod2 in promoting type 1 immunity to Toxoplasma gondii

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Nod2 is required for the regulation of commensal microbiota in the intestine

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Gut commensal bacteria direct a protective immune response against Toxoplasma gondii.

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Artemisone and artemiside control acute and reactivated toxoplasmosis in a murine model

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Gr1(+) inflammatory monocytes are required for mucosal resistance to the pathogen Toxoplasma gondii

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Synergistic effect of Nod1 and Nod2 agonists with toll-like receptor agonists on human dendritic cells to generate interleukin-12 and T helper type 1 cells

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Association of CD4+ T cell-dependent, interferon-gamma-mediated necrosis of the small intestine with genetic susceptibility of mice to peroral infection with Toxoplasma gondii

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Exacerbation of murine ileitis by Toll-like receptor 4 mediated sensing of lipopolysaccharide from commensal Escherichia coli

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Genetic regulation of early survival and cyst number after peroral Toxoplasma gondii infection of A x B/B x A recombinant inbred and B10 congenic mice

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Immune responses associated with early survival after peroral infection with Toxoplasma gondii.

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Epithelial barrier disruption allows nondisease-causing bacteria to initiate and sustain IBD in the IL-10 gene-deficient mouse.

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Quantitative PCR assays for mouse enteric flora reveal strain-dependent differences in composition that are influenced by the microenvironment

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Induction of Nod2 in Myelomonocytic and Intestinal Epithelial Cells via Nuclear Factor-κB Activation

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Genetics and environmental interactions shape the intestinal microbiome to promote inflammatory bowel disease versus mucosal homeostasis

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10.1371/JOURNAL.PONE.0105120

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2014PLoSO...9j5120H

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