Functional AMPA/kainate receptors in human embryonic and foetal central nervous system (Q27867711)

Kainate receptor gene expression in the developing rat brain

PubMed

volume

14

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issue

2

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page(s)

79-85

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cites work

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Modulation of AMPA/kainate receptors by cyclothiazide increases cytoplasmic free Ca2+ and 45Ca2+ uptake in brain neurons

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Developmental regulation of mRNAs encoding rat brain kainate/AMPA receptors: a northern analysis study

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AMPA receptor heterogeneity in rat hippocampal neurons revealed by differential sensitivity to cyclothiazide

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Expression and regulation of kainate and AMPA receptors in uncommitted and committed neural progenitors.

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Blockade of NMDA receptors increases cell death and birth in the developing rat dentate gyrus.

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AMPA receptor development in rat telencephalon: [3H]AMPA binding and western blot studies.

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Quantitative and qualitative changes in AMPA receptor expression during spinal cord development.

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Early functional glutamate receptors in acutely dissociated embryonic raphe cells.

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AMPA elicits long-lasting, partly hypothermia-sensitive calcium responses in acutely dissociated or cultured embryonic brainstem cells.

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Physiological and pathophysiological roles of excitatory amino acids during central nervous system development

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Sensitivity of cultured human embryonic cerebral cortical neurons to excitatory amino acid-induced calcium influx and neurotoxicity

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Differential RNA editing efficiency of AMPA receptor subunit GluR-2 in human brain

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Developmental changes in [3H]kainate binding in human brainstem sites vulnerable to perinatal hypoxia-ischemia.

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Selective modulation of desensitization at AMPA versus kainate receptors by cyclothiazide and concanavalin A.

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Agonist-activated cobalt uptake identifies divalent cation-permeable kainate receptors on neurons and glial cells

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Recognition, adhesion, transmembrane signaling and cell motility in guided neuronal migration

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Human fetal central neurons in culture: voltage- and ligand-gated currents

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Lathyrism: evidence for role of the neuroexcitatory aminoacid BOAA.

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Action potentials, calcium transients and the control of differentiation of excitable cells

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Cortical neurones exhibiting kainate-activated Co2+ uptake are selectively vulnerable to AMPA/kainate receptor-mediated toxicity

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