Elg1 forms an alternative PCNA-interacting RFC complex required to maintain genome stability (Q27935364)

PubMed

13678589

1 December 2016

title

Elg1 forms an alternative PCNA-interacting RFC complex required to maintain genome stability (English)

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PubMed

1 December 2016

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3

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author name string

Pamela Kanellis

series ordinal

1

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Roger Agyei

series ordinal

2

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language of work or name

English

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publication date

16 September 2003

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1 reference

13678589

Chromosome aberrations and cancer

1 December 2016

volume

13

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issue

18

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page(s)

1583-95

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cites work

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Maintenance of genome stability in Saccharomyces cerevisiae

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Regulation of DNA replication fork progression through damaged DNA by the Mec1/Rad53 checkpoint

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ATR homolog Mec1 promotes fork progression, thus averting breaks in replication slow zones.

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Suppression of spontaneous chromosomal rearrangements by S phase checkpoint functions in Saccharomyces cerevisiae

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Caretaker tumour suppressor genes that defend genome integrity

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Telomere dysfunction increases mutation rate and genomic instability

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Multiple pathways cooperate in the suppression of genome instability in Saccharomyces cerevisiae

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The yeast CDK inhibitor Sic1 prevents genomic instability by promoting replication origin licensing in late G(1)

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Deregulated G1-cyclin expression induces genomic instability by preventing efficient pre-RC formation

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Rescue of arrested replication forks by homologous recombination

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Gross chromosomal rearrangements in Saccharomyces cerevisiae replication and recombination defective mutants

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The distribution of the numbers of mutants in bacterial populations

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Characterization of the Five Replication Factor C Genes of Saccharomyces cerevisiae

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Motors and switches: AAA+ machines within the replisome.

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ATP utilization by yeast replication factor C. IV. RFC ATP-binding mutants show defects in DNA replication, DNA repair, and checkpoint regulation

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Mammalian DNA polymerase auxiliary proteins: analysis of replication factor C-catalyzed proliferating cell nuclear antigen loading onto circular double-stranded DNA

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Yeast Rad17/Mec3/Ddc1: a sliding clamp for the DNA damage checkpoint

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Characterization of a mutant strain of Saccharomyces cerevisiae with a deletion of the RAD27 gene, a structural homolog of the RAD2 nucleotide excision repair gene

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A novel mutation avoidance mechanism dependent on S. cerevisiae RAD27 is distinct from DNA mismatch repair

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Chromosomal rearrangements occur in S. cerevisiae rfa1 mutator mutants due to mutagenic lesions processed by double-strand-break repair

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Holliday junctions accumulate in replication mutants via a RecA homolog-independent mechanism

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Recombinational substrates designed to study recombination between unique and repetitive sequences in vivo

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Elevation of sister chromatid exchange in Saccharomyces cerevisiae sgs1 disruptants and the relevance of the disruptants as a system to evaluate mutations in Bloom's syndrome gene

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The DNA replication checkpoint response stabilizes stalled replication forks

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Chl12 (Ctf18) forms a novel replication factor C-related complex and functions redundantly with Rad24 in the DNA replication checkpoint pathway

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Specific DNA replication mutations affect telomere length in Saccharomyces cerevisiae

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Interaction between PCNA and DNA ligase I is critical for joining of Okazaki fragments and long-patch base-excision repair

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DNA polymerase clamp shows little turnover at established replication sites but sequential de novo assembly at adjacent origin clusters

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Suppression of Saccharomyces cerevisiae rad27 null mutant phenotypes by the 5' nuclease domain of Escherichia coli DNA polymerase I.

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A yeast replicative helicase, Dna2 helicase, interacts with yeast FEN-1 nuclease in carrying out its essential function

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Characterization of the Enzymatic Properties of the Yeast Dna2 Helicase/Endonuclease Suggests a New Model for Okazaki Fragment Processing

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A common set of at least 11 functional genes is lost in the majority of NF1 patients with gross deletions

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13678589

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Consolidated OpenCitations Corpus – April 2017