Translin binds to the sequences adjacent to the breakpoints of the TLS and CHOP genes in liposarcomas with translocation t(12;6) (Q28140919)

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H Kanoe

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T Nakayama

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H Murakami

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H Yamamoto

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T Nakamata

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T Tsuboyama

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M Oka

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M Kasai

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M S Sasaki

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T Nakamura

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J Toguchida

A novel gene, Translin, encodes a recombination hotspot binding protein associated with chromosomal translocations

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language of work or name

English

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publication date

23 November 2000

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published in

Oncogene

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volume

19

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issue

50

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page(s)

5821-5

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cites work

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Crossover breakpoint mapping identifies a subtelomeric hotspot for male meiotic recombination

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Translin recognition site sequences flank chromosome translocation breakpoints in alveolar rhabdomyosarcoma cell lines.

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Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma

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A DNA damage repair mechanism is involved in the origin of chromosomal translocations t(4;11) in primary leukemic cells.

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The translin ring specifically recognizes DNA ends at recombination hot spots in the human genome

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Chromosomal translocations in human cancer.

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Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma

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The double-strand-break repair model for recombination

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The RNA-binding protein, TB-RBP, is the mouse homologue of translin, a recombination protein associated with chromosomal translocations

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Chromosome translocation based on illegitimate recombination in human tumors

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Characteristics of genomic breakpoints in TLS-CHOP translocations in liposarcomas suggest the involvement of Translin and topoisomerase II in the process of translocation

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Cloning and sequence analysis of four t(9;11) therapy-related leukemia breakpoints

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inferred from DOI database lookup

7 January 2021

based on heuristic

Mechanism of action of eukaryotic topoisomerase II and drugs targeted to the enzyme

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Repair and recombination. How to make ends meet

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