Aspirin insensitive eicosanoid biosynthesis in cardiovascular disease (Q28165894)

Europe PubMed Central

title

Aspirin insensitive eicosanoid biosynthesis in cardiovascular disease (English)

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cardiovascular disease

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1

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Paola Patrignani

series ordinal

1

object named as

Paola Patrignani

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Europe PubMed Central

PubMed ID

14592549

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A14592549

full work available at URL

27 March 2024

language of work or name

English

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publication date

15 June 2003

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https://api.elsevier.com/content/article/PII:S0049384803003827?httpAccept=text/xml

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https://api.crossref.org/works/10.1016/S0049-3848(03)00382-7

https://api.elsevier.com/content/article/PII:S0049384803003827?httpAccept=text/plain

27 March 2024

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https://api.crossref.org/works/10.1016/S0049-3848(03)00382-7

27 March 2024

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110

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issue

5-6

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page(s)

281-6

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cites work

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Platelet activation in unstable coronary disease

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Differential Suppression of Thromboxane Biosynthesis by Indobufen and Aspirin in Patients With Unstable Angina

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Oxidant stress and aspirin-insensitive thromboxane biosynthesis in severe unstable angina

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Modulation of aspirin-insensitive eicosanoid biosynthesis by 6-methylprednisolone in unstable angina

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Increased urinary leukotriene excretion in patients with cardiac ischemia. In vivo evidence for 5-lipoxygenase activation

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Angioplasty triggers intracoronary leukotrienes and lipoxin A4. Impact of aspirin therapy

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Mechanisms of platelet activation: thromboxane A2 as an amplifying signal for other agonists

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Coronary arterial smooth muscle contraction by a substance released from platelets: evidence that it is thromboxane A2

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Overexpression of functionally coupled cyclooxygenase-2 and prostaglandin E synthase in symptomatic atherosclerotic plaques as a basis of prostaglandin E(2)-dependent plaque instability

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The structural basis of aspirin activity inferred from the crystal structure of inactivated prostaglandin H2 synthase

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Selective cumulative inhibition of platelet thromboxane production by low-dose aspirin in healthy subjects

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Cyclooxygenase-2 expression is induced during human megakaryopoiesis and characterizes newly formed platelets

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Cyclooxygenases-1 and −2 of Endothelial Cells Utilize Exogenous or Endogenous Arachidonic Acid for Transcellular Production of Thromboxane

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Aspirin-resistant thromboxane biosynthesis and the risk of myocardial infarction, stroke, or cardiovascular death in patients at high risk for cardiovascular events

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Local amplification of platelet function by 8-Epi prostaglandin F2alpha is not mediated by thromboxane receptor isoforms

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Leukotrienes: Mediators of Immediate Hypersensitivity Reactions and Inflammation

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Characterization of the human cysteinyl leukotriene 2 receptor

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COX-2 is not involved in thromboxane biosynthesis by activated human platelets

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27 March 2024

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