Aspirin induces apoptosis through mitochondrial cytochrome c release (Q28174710)

2

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M Dalmau

3

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B Bellosillo

4

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G Pons

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J Gil

Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells

6

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language of work or name

English

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publication date

1 September 2000

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published in

FEBS Letters

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volume

480

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issue

2-3

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page(s)

193-6

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cites work

1 reference

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Aspirin induces cell death and caspase-dependent phosphatidylserine externalization in HT-29 human colon adenocarcinoma cells

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Nonsteroidal antiinflammatory drugs cause apoptosis and induce cyclooxygenases in chicken embryo fibroblasts

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Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation.

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The effects of sulindac on colorectal proliferation and apoptosis in familial adenomatous polyposis.

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Caspases: enemies within

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Apoptotic pathways: the roads to ruin

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Molecular characterization of mitochondrial apoptosis-inducing factor

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Mitochondria and apoptosis

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Mitochondrial control of cell death

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Presence of a pre-apoptotic complex of pro-caspase-3, Hsp60 and Hsp10 in the mitochondrial fraction of jurkat cells

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Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

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Release of cytochrome c from liver mitochondria during permeability transition.

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Disruption of the outer mitochondrial membrane as a result of large amplitude swelling: the impact of irreversible permeability transition.

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Role of the mitochondrial permeability transition in salicylate toxicity to cultured rat hepatocytes: implications for the pathogenesis of Reye's syndrome

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Mechanisms underlying nonsteroidal antiinflammatory drug-mediated apoptosis

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Sulindac inhibits activation of the NF-kappaB pathway

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Inhibition of nuclear factor kappa B and induction of apoptosis in T-lymphocytes by sulfasalazine

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PPARdelta is an APC-regulated target of nonsteroidal anti-inflammatory drugs

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Activation of apoptotic caspase-3 and nitric oxide synthase-2 in gastric mucosal injury induced by indomethacin

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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Cytochrome c: can't live with it--can't live without it.

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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Cytochrome c release and caspase activation in hydrogen peroxide- and tributyltin-induced apoptosis

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

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Distinct stages of cytochrome c release from mitochondria: evidence for a feedback amplification loop linking caspase activation to mitochondrial dysfunction in genotoxic stress induced apoptosis

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The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

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Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

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Salicylate and mitochondrial injury in Reye's syndrome

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Alterations in cellular adhesion and apoptosis in epithelial cells overexpressing prostaglandin endoperoxide synthase 2

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7 January 2021

Identifiers

DOI

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Fatcat ID

release_wljaxexepvcvtgitbfmunvfzza

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/wljaxexepvcvtgitbfmunvfzza