Modulation of age at onset in Huntington's disease and spinocerebellar ataxia type 2 patients originated from eastern India (Q28181424)

inferred from title

author name string

Biswanath Chattopadhyay

1

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Subho Ghosh

2

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Prasanta K Gangopadhyay

3

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Shaymal K Das

4

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T Roy

5

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Krishna K Sinha

6

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Dilip K Jha

7

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Subhash C Mukherjee

8

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Ambar Chakraborty

9

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Bhim S Singhal

10

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Anup K Bhattacharya

11

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Nitai P Bhattacharyya

CAG repeat length in RAI1 is associated with age at onset variability in spinocerebellar ataxia type 2 (SCA2)

12

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language of work or name

English

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publication date

17 July 2003

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published in

Neuroscience Letters

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volume

345

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issue

2

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page(s)

93-6

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cites work

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The Gln-Ala repeat transcriptional activator CA150 interacts with huntingtin: neuropathologic and genetic evidence for a role in Huntington's disease pathogenesis

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Evidence for the GluR6 gene associated with younger onset age of Huntington's disease

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Analysis of CAG and CCG repeats in Huntingtin gene among HD patients and normal populations of India

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Genotypes at the GluR6 kainate receptor locus are associated with variation in the age of onset of Huntington disease

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DNA haplotype analysis of Huntington disease reveals clues to the origins and mechanisms of CAG expansion and reasons for geographic variations of prevalence

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Onset and pre-onset studies to define the Huntington's disease natural history.

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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