Transgenic incorporation of skeletal TnT into cardiac myofilaments blunts PKC-mediated depression of force (Q28200096)

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M Chandra

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Q Huang

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J Jin

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R J Solaro

American Journal of Physiology Heart and Circulatory Physiology

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language of work or name

English

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publication date

March 2001

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published in

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volume

280

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issue

3

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page(s)

H1011-8

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cites work

Expression of protein kinase C beta in the heart causes hypertrophy in adult mice and sudden death in neonates.

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The N-terminal region of troponin T is essential for the maximal activation of rat cardiac myofilaments

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Protein kinase C enhances myosin light-chain kinase effects on force development and ATPase activity in rat single skinned cardiac cells

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M2-specific muscarinic cholinergic receptor-mediated inhibition of cardiac regulatory protein phosphorylation

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Fast skeletal muscle troponin T increases the cooperativity of transgenic mouse cardiac muscle contraction.

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Three alternatively spliced mouse slow skeletal muscle troponin T isoforms: conserved primary structure and regulated expression during postnatal development

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Complete nucleotide sequence and structural organization of rat cardiac troponin T gene

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Expression of cDNAs encoding mouse cardiac troponin T isoforms: characterization of a large sample of independent clones

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Localization of protein kinases by anchoring proteins: a theme in signal transduction

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Identification of intracellular receptor proteins for activated protein kinase C

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Cardiac troponin I mutants. Phosphorylation by protein kinases C and A and regulation of Ca(2+)-stimulated MgATPase of reconstituted actomyosin S-1

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Different myofilament nearest-neighbor interactions have distinctive effects on contractile behavior

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Altered interactions among thin filament proteins modulate cardiac function

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In vivo phosphorylation of cardiac troponin I by protein kinase Cbeta2 decreases cardiomyocyte calcium responsiveness and contractility in transgenic mouse hearts

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Thin filament-mediated regulation of cardiac contraction

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Conformational modulation of troponin T by configuration of the NH2-terminal variable region and functional effects

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Relation between crossbridge structure and actomyosin ATPase activity in rat heart

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