P75 interacts with the Nogo receptor as a co-receptor for Nogo, MAG and OMgp (Q28214328)

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Jieun A Kim

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Rajeev Sivasankaran

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Rosalind Segal

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Zhigang He

Identification of a receptor mediating Nogo-66 inhibition of axonal regeneration

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language of work or name

English

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publication date

7 November 2002

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published in

Nature

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volume

420

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page(s)

74-8

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issue

6911

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cites work

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Oligodendrocyte-myelin glycoprotein is a Nogo receptor ligand that inhibits neurite outgrowth

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Myelin-associated glycoprotein as a functional ligand for the Nogo-66 receptor

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Biochemical and functional interactions between the neurotrophin receptors trk and p75NTR

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Identification of the Nogo inhibitor of axon regeneration as a Reticulon protein

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Calcium sensitization of smooth muscle mediated by a Rho-associated protein kinase in hypertension

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Regulation of the small GTP-binding protein Rho by cell adhesion and the cytoskeleton

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Myelin-associated glycoprotein interacts with the Nogo66 receptor to inhibit neurite outgrowth

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Signalling through the neurotrophin receptor p75NTR.

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The uniqueness of being a neurotrophin receptor

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The p75 receptor transduces the signal from myelin-associated glycoprotein to Rho.

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Plexins are a large family of receptors for transmembrane, secreted, and GPI-anchored semaphorins in vertebrates

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Targeted mutation of the gene encoding the low affinity NGF receptor p75 leads to deficits in the peripheral sensory nervous system

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A mouse serine/threonine kinase homologous to C. elegans UNC51 functions in parallel fiber formation of cerebellar granule neurons

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Neurotrophin binding to the p75 receptor modulates Rho activity and axonal outgrowth

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Developmental and regional expression of beta-nerve growth factor receptor mRNA in the chick and rat.

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Alkaline phosphatase fusion proteins for molecular characterization and cloning of receptors and their ligands

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Neuropilin is a receptor for the axonal chemorepellent Semaphorin III

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Myelination in rat brain: method of myelin isolation.

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Effects of brain-derived neurotrophic factor on optic axon branching and remodelling in vivo

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Myelin-associated glycoprotein interacts with ganglioside GT1b. A mechanism for neurite outgrowth inhibition

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Inactivation of Rho signaling pathway promotes CNS axon regeneration

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