PGE(2) in the regulation of programmed erythrocyte death (Q28218548)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15746942%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

title

PGE(2) in the regulation of programmed erythrocyte death (English)

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9 January 2020

6

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9 January 2020

author name string

P A Lang

1

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9 January 2020

D S Kempe

2

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9 January 2020

S Myssina

3

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9 January 2020

V Tanneur

4

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9 January 2020

C Birka

5

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9 January 2020

F Lang

7

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9 January 2020

T Wieder

8

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9 January 2020

S M Huber

9

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9 January 2020

language of work or name

English

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publication date

1 May 2005

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Cell Death & Differentiation

9 January 2020

published in

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9 January 2020

volume

12

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9 January 2020

issue

5

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9 January 2020

page(s)

415-428

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A receptor for phosphatidylserine-specific clearance of apoptotic cells

9 January 2020

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Programmed cell death in mature erythrocytes: a model for investigating death effector pathways operating in the absence of mitochondria

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Role of Ca2+-activated K+ channels in human erythrocyte apoptosis

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A necessary role for reduced intracellular potassium during the DNA degradation phase of apoptosis

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Caspase 3 regulates phosphatidylserine externalization and phagocytosis of oxidatively stressed erythrocytes

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Caspase 3-mediated proteolysis of the N-terminal cytoplasmic domain of the human erythroid anion exchanger 1 (band 3).

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Oxidation and erythrocyte senescence

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7 January 2021

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Identifiers

DOI

10.1038/SJ.CDD.4401561

2 references

Consolidated OpenCitations Corpus – April 2017

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15746942%20AND%20SRC:MED&resulttype=core&format=json

9 January 2020

1 reference

Consolidated OpenCitations Corpus – April 2017

2 references

Consolidated OpenCitations Corpus – April 2017