Six and Eya expression during human somitogenesis and MyoD gene family activation (Q28219080)

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Muriel Durand

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Soledad Lopez

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Laurence Suel

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Josiane Demignon

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Charles Thornton

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Hidenori Ozaki

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Kyoshi Kawakami

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Patrick Barbet

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Jacques S Beckmann

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Pascal Maire

Journal of Muscle Research & Cell Motility

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language of work or name

English

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publication date

2002

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published in

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volume

23

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issue

3

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page(s)

255-64

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255-264

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The eye-specification proteins So and Eya form a complex and regulate multiple steps in Drosophila eye development

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Eya1-deficient mice lack ears and kidneys and show abnormal apoptosis of organ primordia

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Six family genes--structure and function as transcription factors and their roles in development

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Drosophila homolog of the myotonic dystrophy-associated gene, SIX5, is required for muscle and gonad development.

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Six4, a putative myogenin gene regulator, is not essential for mouse embryonal development.

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The alpha subunits of Gz and Gi interact with the eyes absent transcription cofactor Eya2, preventing its interaction with the six class of homeodomain-containing proteins

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Cooperation of six and eya in activation of their target genes through nuclear translocation of Eya

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MyoD, myogenin independent differentiation of primordial myoblasts in mouse somites

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Immaturity of muscle fibers in the congenital form of myotonic dystrophy: its consequences and its origin

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Mouse Eya genes are expressed during limb tendon development and encode a transcriptional activation function

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Expression of genes (CAPN3, SGCA, SGCB, and TTN) involved in progressive muscular dystrophies during early human development

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Eyeless initiates the expression of both sine oculis and eyes absent during Drosophila compound eye development.

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sine oculis is a homeobox gene required for Drosophila visual system development

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alpha11beta1 integrin is a receptor for interstitial collagens involved in cell migration and collagen reorganization on mesenchymal nonmuscle cells

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Multiple roles of the eyes absent gene in Drosophila

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Trinucleotide repeat expansion at the myotonic dystrophy locus reduces expression of DMAHP.

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Mouse Eya homologues of the Drosophila eyes absent gene require Pax6 for expression in lens and nasal placode

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