Stable tubule only polypeptides (STOP) proteins co-aggregate with spheroid neurofilaments in amyotrophic lateral sclerosis (Q28235829)

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A Bocquet

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F Dubas

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A Barthelaix

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J Eyer

Journal of Neuropathology & Experimental Neurology

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language of work or name

English

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publication date

December 2003

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published in

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volume

62

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issue

12

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page(s)

1211-9

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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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Pathogenesis of two axonopathies does not require axonal neurofilaments.

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Purification and assay of a 145-kDa protein (STOP145) with microtubule-stabilizing and motility behavior

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Cloning, expression, and properties of the microtubule-stabilizing protein STOP

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Nonneuronal isoforms of STOP protein are responsible for microtubule cold stability in mammalian fibroblasts

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STOP proteins are responsible for the high degree of microtubule stabilization observed in neuronal cells

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Ca(2+)-calmodulin regulated effectors of microtubule stability in neuronal tissues

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Monoclonal antibody to microtubule-associated STOP protein: affinity purification of neuronal STOP activity and comparison of antigen with activity in neuronal and nonneuronal cell extracts.

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Influence of the phosphorylation state of neurofilament proteins on the interactions between purified filaments in vitro

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Identification of novel bifunctional calmodulin-binding and microtubule-stabilizing motifs in STOP proteins.

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Galectin-1 Is a Component of Neurofilamentous Lesions in Sporadic and Familial Amyotrophic Lateral Sclerosis

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Deletions of the heavy neurofilament subunit tail in amyotrophic lateral sclerosis

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