Soluble endoglin contributes to the pathogenesis of preeclampsia (Q28244053)

Patricia A D'Amore

13

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Towia A Libermann

Towia A Libermann

10

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S Ananth Karumanchi

19

S Ananth Karumanchi

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Roberto Romero

Roberto Romero

16

1 reference

ORCID Public Data File 2021

author name string

Shivalingappa Venkatesha

1

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Chun Lam

3

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Jun-ichi Hanai

4

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Tadanori Mammoto

5

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Yeon M Kim

6

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Yuval Bdolah

7

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Kee-Hak Lim

8

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Hai-Tao Yuan

9

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Isaac E Stillman

11

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Drucilla Roberts

12

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Franklin H Epstein

14

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Frank W Sellke

15

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Vikas P Sukhatme

17

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Michelle Letarte

Syndrome of hemolysis, elevated liver enzymes, and low platelet count: a severe consequence of hypertension in pregnancy

18

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language of work or name

English

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publication date

June 2006

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published in

Nature Medicine

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volume

12

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issue

6

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page(s)

642-9

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cites work

1 reference

7 January 2021

Preeclampsia: an endothelial cell disorder

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7 January 2021

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

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Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets syndrome

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Elevated placental soluble vascular endothelial growth factor receptor-1 inhibits angiogenesis in preeclampsia.

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Evidence supporting a role for blockade of the vascular endothelial growth factor system in the pathophysiology of preeclampsia. Young Investigator Award.

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Longitudinal serum concentrations of placental growth factor: evidence for abnormal placental angiogenesis in pathologic pregnancies

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Circulating angiogenic factors and the risk of preeclampsia

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Plasma soluble vascular endothelial growth factor receptor-1 concentration is elevated prior to the clinical diagnosis of pre-eclampsia

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Maternal serum sFlt1 concentration is an early and reliable predictive marker of preeclampsia

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Clinical significance, prevalence, and natural history of thrombocytopenia in pregnancy-induced hypertension

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Identification of distinct epitopes of endoglin, an RGD-containing glycoprotein of endothelial cells, leukemic cells, and syncytiotrophoblasts

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7 January 2021

Localization of endoglin, a transforming growth factor-beta binding protein, and of CD44 and integrins in placenta during the first trimester of pregnancy

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7 January 2021

Endoglin, a TGF-beta binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1

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A murine model of hereditary hemorrhagic telangiectasia

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7 January 2021

Defective angiogenesis in mice lacking endoglin

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A role for endoglin in coupling eNOS activity and regulating vascular tone revealed in hereditary hemorrhagic telangiectasia

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Phosphorylation of the endothelial nitric oxide synthase at ser-1177 is required for VEGF-induced endothelial cell migration

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Dynamic activation of endothelial nitric oxide synthase by Hsp90.

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Capillary permeability and extracellular fluid volumes in pregnancy-induced hypertension

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Molecular regulation of the bovine endothelial cell nitric oxide synthase by transforming growth factor-beta 1.

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Smad2 mediates transforming growth factor-beta induction of endothelial nitric oxide synthase expression

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Latest advances in understanding preeclampsia

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Plasma levels of soluble CD105 correlate with metastasis in patients with breast cancer

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The shedding of betaglycan is regulated by pervanadate and mediated by membrane type matrix metalloprotease-1.

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Transforming growth factor-beta1 (TGF-beta1) in plasma is associated with preeclampsia risk in Peruvian women with systemic inflammation

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7 January 2021

Transforming growth factor-beta 1 does not relate to hypertension in pre-eclampsia.

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7 January 2021

Endoglin is an accessory protein that interacts with the signaling receptor complex of multiple members of the transforming growth factor-beta superfamily

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7 January 2021

Elevated blood pressures in mice lacking endothelial nitric oxide synthase.

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7 January 2021

Nitric oxide dysfunction in the pathophysiology of preeclampsia

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Nitric oxide production contributes to the angiogenic properties of vascular endothelial growth factor in human endothelial cells

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Constitutive eNOS-derived nitric oxide is a determinant of endothelial junctional integrity

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Transforming growth factor-beta(1) restores antiplatelet function of endothelial cells exposed to anoxia-reoxygenation injury.

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Effect of growth factors on human vascular endothelial cell prostacyclin production

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Vascular endothelial growth factor signals endothelial cell production of nitric oxide and prostacyclin through flk-1/KDR activation of c-Src

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7 January 2021

Prostacyclin and thromboxane changes predating clinical onset of preeclampsia: a multicenter prospective study

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Pericyte production of cell-associated VEGF is differentiation-dependent and is associated with endothelial survival

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Human cytotrophoblasts adopt a vascular phenotype as they differentiate. A strategy for successful endovascular invasion?

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Preeclampsia is associated with failure of human cytotrophoblasts to mimic a vascular adhesion phenotype. One cause of defective endovascular invasion in this syndrome?

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Endoglin regulates trophoblast differentiation along the invasive pathway in human placental villous explants

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Endoglin (CD105): a target for anti-angiogenetic cancer therapy

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VEGF-Trap: a VEGF blocker with potent antitumor effects

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Comparative evaluation of the antitumor activity of antiangiogenic proteins delivered by gene transfer

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c-Ski acts as a transcriptional co-repressor in transforming growth factor-beta signaling through interaction with smads

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7 January 2021