Hypoxia induces type II NOS gene expression in pulmonary artery endothelial cells via HIF-1 (Q28263501)

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9 November 2019

title

Hypoxia induces type II NOS gene expression in pulmonary artery endothelial cells via HIF-1 (English)

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9 November 2019

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2

G L Semenza

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9 November 2019

author name string

L A Palmer

series ordinal

1

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9 November 2019

M H Stoler

series ordinal

3

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9 November 2019

R A Johns

series ordinal

4

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9 November 2019

language of work or name

English

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publication date

1 February 1998

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American Journal of Physiology

9 November 2019

published in

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9 November 2019

volume

274

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9 November 2019

issue

2

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9 November 2019

page(s)

L212-9

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In vivo expression of mRNAs encoding hypoxia-inducible factor 1

9 November 2019

cites work

1 reference

12 October 2022

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Promoter of the mouse gene encoding calcium-independent nitric oxide synthase confers inducibility by interferon gamma and bacterial lipopolysaccharide

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The transcription factors ATF-1 and CREB-1 bind constitutively to the hypoxia-inducible factor-1 (HIF-1) DNA recognition site

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Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

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Increased gene expression for VEGF and the VEGF receptors KDR/Flk and Flt in lungs exposed to acute or to chronic hypoxia. Modulation of gene expression by nitric oxide

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Hypoxia-inducible Factor-1 Mediates Transcriptional Activation of the Heme Oxygenase-1 Gene in Response to Hypoxia

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The structure of the promoter region for rat inducible nitric oxide synthase gene

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Hypoxic Regulation of Lactate Dehydrogenase A

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Endothelium-derived relaxing factor activity in rat lung during hypoxic pulmonary vascular remodeling.

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NG-monomethyl L-arginine inhibits endothelium-derived relaxing factor-stimulated cyclic GMP accumulation in cocultures of endothelial and vascular smooth muscle cells by an action specific to the endothelial cell

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Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

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Transcriptional regulation of the rat vascular endothelial growth factor gene by hypoxia

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Prolonged in vivo hypoxia enhances nitric oxide synthase type I and type III gene expression in adult rat lung

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General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

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Activation of hypoxia-inducible transcription factor depends primarily upon redox-sensitive stabilization of its alpha subunit

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A hypoxia-responsive element mediates a novel pathway of activation of the inducible nitric oxide synthase promoter

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Hypoxia induces endothelin gene expression and secretion in cultured human endothelium

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Upregulation of nitric oxide synthase correlates temporally with onset of pulmonary vascular remodeling in the hypoxic rat.

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Regulation of angiogenic growth factor expression by hypoxia, transition metals, and chelating agents

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Oxygen-regulated control elements in the phosphoglycerate kinase 1 and lactate dehydrogenase A genes: similarities with the erythropoietin 3' enhancer

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Recombinant genomes which express chloramphenicol acetyltransferase in mammalian cells

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A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation

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Hypoxia Response Elements in the Aldolase A, Enolase 1, and Lactate Dehydrogenase A Gene Promoters Contain Essential Binding Sites for Hypoxia-inducible Factor 1

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12 October 2022

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Effect of altered redox states on expression and DNA-binding activity of hypoxia-inducible factor 1.

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Oxygen tension regulates the expression of the platelet-derived growth factor-B chain gene in human endothelial cells

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Enhanced endothelin-1 and endothelin receptor gene expression in chronic hypoxia

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Purification and characterization of hypoxia-inducible factor 1

1 reference

12 October 2022

https://opencitations.net/index/coci/api/v1/references/10.1152/AJPLUNG.1998.274.2.L212

Identifiers

DOI

10.1152/AJPLUNG.1998.274.2.L212

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9486205%20AND%20SRC:MED&resulttype=core&format=json

OpenCitations bibliographic resource ID

9 November 2019

4849219

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4849219

2 references

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4849219