Loss of RAGE in pulmonary fibrosis: molecular relations to functional changes in pulmonary cell types (Q28276929)

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6 January 2020

title

Loss of RAGE in pulmonary fibrosis: molecular relations to functional changes in pulmonary cell types (English)

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6 January 2020

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4

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Klaus T Preissner

1 reference

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6 January 2020

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Oliver Eickelberg

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6 January 2020

author name string

Markus A Queisser

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6 January 2020

Fotini M Kouri

2

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6 January 2020

Melanie Königshoff

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Uwe Schubert

5

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language of work or name

English

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publication date

17 April 2008

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American Journal of Respiratory Cell and Molecular Biology

6 January 2020

published in

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volume

39

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page(s)

337-345

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issue

3

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Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

6 January 2020

cites work

1 reference

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Idiopathic pulmonary fibrosis: an epithelial/fibroblastic cross-talk disorder

21 January 2018

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Mechanisms of pulmonary fibrosis

21 January 2018

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Understanding RAGE, the receptor for advanced glycation end products

21 January 2018

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Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

21 January 2018

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Diabetes-associated sustained activation of the transcription factor nuclear factor-kappaB

21 January 2018

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Promotion of cell adherence and spreading: a novel function of RAGE, the highly selective differentiation marker of human alveolar epithelial type I cells.

21 January 2018

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The pattern recognition receptor (RAGE) is a counterreceptor for leukocyte integrins: a novel pathway for inflammatory cell recruitment

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Contribution of CFTR to apical-basolateral fluid transport in cultured human alveolar epithelial type II cells

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Caveolin-1: a critical regulator of lung fibrosis in idiopathic pulmonary fibrosis

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Increased bleomycin-induced lung injury in mice deficient in the transcription factor T-bet.

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Synaptopodin orchestrates actin organization and cell motility via regulation of RhoA signalling

21 January 2018

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Purification and characterization of mouse soluble receptor for advanced glycation end products (sRAGE)

21 January 2018

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The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-alpha through nuclear factor-kappa B, and by 17beta-estradiol through Sp-1 in human vascular endothelial cells

21 January 2018

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Expression of a tumor necrosis factor-alpha transgene in murine lung causes lymphocytic and fibrosing alveolitis. A mouse model of progressive pulmonary fibrosis

21 January 2018

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Fibroblasts from idiopathic pulmonary fibrosis and normal lungs differ in growth rate, apoptosis, and tissue inhibitor of metalloproteinases expression

21 January 2018

1 reference

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Enhanced migration of fibroblasts derived from lungs with fibrotic lesions

21 January 2018

1 reference

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Fibroblasts isolated from normal lungs and those with idiopathic pulmonary fibrosis differ in interleukin-6/gp130-mediated cell signaling and proliferation

21 January 2018

1 reference

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Down-regulation of the receptor for advanced glycation end-products (RAGE) supports non-small cell lung carcinoma

21 January 2018

1 reference

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Proliferative stimulus of lung fibroblasts on lung cancer cells is impaired by the receptor for advanced glycation end-products

21 January 2018

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The role of the receptor for advanced glycation end-products in lung fibrosis

21 January 2018

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21 January 2018

Identifiers

DOI

10.1165/RCMB.2007-0244OC

2 references

Consolidated OpenCitations Corpus – April 2017

2661754

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6 January 2020

2661754

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Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

2 references

2661754