Endoplasmic reticulum stress and induction of the unfolded protein response in human sporadic amyotrophic lateral sclerosis (Q28277961)

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6 January 2020

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29 December 2023

https://doi.org/10.1016/j.nbd.2020.105232

http://retractionwatch.com/2017/09/11/authors-withdraw-study-citing-accidentally-duplicated-images/

title

Endoplasmic reticulum stress and induction of the unfolded protein response in human sporadic amyotrophic lateral sclerosis (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18440237%20AND%20SRC:MED&resulttype=core&format=json

amyotrophic lateral sclerosis

6 January 2020

main subject

endoplasmic reticulum

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author

Julie D Atkin

1

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6 January 2020

Adam Walker

3

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6 January 2020

Catriona A. McLean

4

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6 January 2020

2

Manal A Farg

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6 January 2020

author name string

Doris Tomas

5

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6 January 2020

Malcolm K Horne

6

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6 January 2020

language of work or name

English

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publication date

6 March 2008

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6 January 2020

published in

Neurobiology of Disease

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6 January 2020

volume

30

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6 January 2020

page(s)

400-407

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6 January 2020

issue

3

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Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

6 January 2020

cites work

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S-nitrosylated protein-disulphide isomerase links protein misfolding to neurodegeneration

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Ubiquitin-immunoreactive intraneuronal inclusions in amyotrophic lateral sclerosis. Morphology, distribution, and specificity.

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Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Abeta-induced cell death

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ALS: a disease of motor neurons and their nonneuronal neighbors

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Impaired extracellular secretion of mutant superoxide dismutase 1 associates with neurotoxicity in familial amyotrophic lateral sclerosis

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Overexpressed protein disulfide isomerase in brains of patients with sporadic Creutzfeldt-Jakob disease

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Cu/Zn superoxide dismutase-like immunoreactivity in Lewy body-like inclusions of sporadic amyotrophic lateral sclerosis

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Proteins of the PDI family: unpredicted non-ER locations and functions

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Orchestrating the unfolded protein response in health and disease

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A potential role for the p75 low-affinity neurotrophin receptor in spinal motor neuron degeneration in murine and human amyotrophic lateral sclerosis

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Amyotrophic lateral sclerosis is a distal axonopathy: evidence in mice and man

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Chromogranin-mediated secretion of mutant superoxide dismutase proteins linked to amyotrophic lateral sclerosis

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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The Golgi apparatus of motor neurons in amyotrophic lateral sclerosis.

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Spinal cord endoplasmic reticulum stress associated with a microsomal accumulation of mutant superoxide dismutase-1 in an ALS model

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Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

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Aggresomes: a cellular response to misfolded proteins

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Induction of the unfolded protein response in familial amyotrophic lateral sclerosis and association of protein-disulfide isomerase with superoxide dismutase 1.

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Conversion to the amyotrophic lateral sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria

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Identifiers

DOI

10.1016/J.NBD.2008.02.009

2 references

Consolidated OpenCitations Corpus – April 2017

1712494

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18440237%20AND%20SRC:MED&resulttype=core&format=json

6 January 2020

1712494

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Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

2 references

1712494