GAS5, a non-protein-coding RNA, controls apoptosis and is downregulated in breast cancer (Q28296081)

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M R Pickard

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V L Hedge

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G T Williams

Regulation and expression of a growth arrest-specific gene (gas5) during growth, differentiation, and development

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language of work or name

English

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publication date

15 January 2009

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published in

Oncogene

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volume

28

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issue

2

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page(s)

195-208

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cites work

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SnoRNA U50 is a candidate tumor-suppressor gene at 6q14.3 with a mutation associated with clinically significant prostate cancer

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DAP kinase links the control of apoptosis to metastasis

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The snoRNA HBII-52 regulates alternative splicing of the serotonin receptor 2C

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Relevance of breast cancer cell lines as models for breast tumours: an update

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Expression of a defective mouse mammary tumor virus envelope glycoprotein precursor which binds stably to GRP78 within the lumen of the endoplasmic reticulum is associated with decreased glucocorticoid-induced apoptosis in mouse lymphoma cells

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Cell-specific induction of apoptosis by microinjection of cytochrome c. Bcl-xL has activity independent of cytochrome c release

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p53-dependent apoptosis modulates the cytotoxicity of anticancer agents

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Growth arrest in human T-cells is controlled by the non-coding RNA growth-arrest-specific transcript 5 (GAS5).

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Candidate tumor suppressor LUCA-15/RBM5/H37 modulates expression of apoptosis and cell cycle genes

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Functional expression cloning reveals a central role for the receptor for activated protein kinase C 1 (RACK1) in T cell apoptosis

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Regulation of apoptosis by fau revealed by functional expression cloning and antisense expression

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Functional expression cloning reveals proapoptotic role for protein phosphatase 4

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Candidate tumour suppressor LUCA-15 can regulate multiple apoptotic pathways

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The gas5 gene is disrupted by a frameshift mutation within its longest open reading frame in several inbred mouse strains and maps to murine chromosome 1.

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The GAS5 (growth arrest-specific transcript 5) gene fuses to BCL6 as a result of t(1;3)(q25;q27) in a patient with B-cell lymphoma

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The gas 5 gene shows four alternative splicing patterns without coding for a protein

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The toxic responses induced by okadaic acid involve processing of multiple caspase isoforms.

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BID regulation by p53 contributes to chemosensitivity

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Genes specifically expressed at growth arrest of mammalian cells

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High-resolution genomic profiling reveals association of chromosomal aberrations on 1q and 16p with histologic and genetic subgroups of invasive breast cancer.

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LUCA-15-encoded sequence variants regulate CD95-mediated apoptosis

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Intronic U50 small-nucleolar-RNA (snoRNA) host gene of no protein-coding potential is mapped at the chromosome breakpoint t(3;6)(q27;q15) of human B-cell lymphoma

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Breast cancer cells response to the antineoplastic agents cisplatin, carboplatin, and doxorubicin at the mRNA expression levels of distinct apoptosis-related genes, including the new member, BCL2L12.

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Radiation-induced deletions in the 5' end region of Notch1 lead to the formation of truncated proteins and are involved in the development of mouse thymic lymphomas.

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Apoptosis in toremifene-induced growth inhibition of human breast cancer cells in vivo and in vitro

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The use of gene function to identify the rate-limiting steps controlling cell fate

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Molecular regulation of apoptosis: genetic controls on cell death.

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