Heparin inhibits c-fos and c-myc mRNA expression in vascular smooth muscle cells (Q28329014)

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T C Wright

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B L Caleb

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M J Karnovsky

Molecular Biology of the Cell

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language of work or name

English

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publication date

April 1990

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published in

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volume

1

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issue

5

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page(s)

435-43

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cites work

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Platelet-derived growth factor and heparin-like glycosaminoglycans regulate thrombospondin synthesis and deposition in the matrix by smooth muscle cells

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Heparin regulates the collagen phenotype of vascular smooth muscle cells: induced synthesis of an Mr 60,000 collagen

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Interferon regulates c-myc gene expression in Daudi cells at the post-transcriptional level

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Inducible production of c-fos antisense RNA inhibits 3T3 cell proliferation

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An antiproliferative heparan sulfate species produced by postconfluent smooth muscle cells

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Heparin selectively inhibits a protein kinase C-dependent mechanism of cell cycle progression in calf aortic smooth muscle cells

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Heparin suppresses the induction of c-fos and c-myc mRNA in murine fibroblasts by selective inhibition of a protein kinase C-dependent pathway

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Induction of c-fos and c-myc mRNA by epidermal growth factor or calcium ionophore is cAMP dependent

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Inhibitory effects of interferon on the expression of genes regulated by platelet-derived growth factor

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Heparin modulates the secretion of a major excreted protein-like molecule by vascular smooth muscle cells

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Expression of apolipoprotein E by cultured vascular smooth muscle cells is controlled by growth state

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Heparin inhibits proliferation of fetal vascular smooth muscle cells in the absence of platelet-derived growth factor

2 June 2018

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12 December 2020

Regulation of oncogene expression in cultured aortic smooth muscle cells. Post-transcriptional control of c-myc mRNA

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Heparin inhibition of smooth muscle cell proliferation: a cellular site of action

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TGF-beta inhibition of endothelial cell proliferation: alteration of EGF binding and EGF-induced growth-regulatory (competence) gene expression

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Antiproliferative effects of heparin on vascular smooth muscle cells are reversed by epidermal growth factor

1 reference

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A c-myc antisense oligodeoxynucleotide inhibits entry into S phase but not progress from G0 to G1

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The Fos complex and Fos-related antigens recognize sequence elements that contain AP-1 binding sites

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Human arterial smooth muscle cells in culture. Effects of platelet-derived growth factor and heparin on growth in vitro

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Growth factors regulate transin gene expression by c-fos-dependent and c-fos-independent pathways

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Decreased susceptibility of cultured smooth muscle cells from SHR rats to growth inhibition by heparin

1 reference

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Heparin prevents vascular smooth muscle cell progression through the G1 phase of the cell cycle

1 reference

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Negative regulation of serum-responsive enhancer elements

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Suppression by heparin of smooth muscle cell proliferation in injured arteries

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Inhibition of rat arterial smooth muscle cell proliferation by heparin. In vivo studies with anticoagulant and nonanticoagulant heparin

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Inhibition of rat arterial smooth muscle cell proliferation by heparin. II. In vitro studies

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Inhibition of vascular smooth muscle cell growth by endothelial cell-derived heparin. Possible role of a platelet endoglycosidase

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Effect of heparin on vascular smooth muscle cells. I. Cell metabolism

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The antiproliferative effect of interferon and the mitogenic activity of growth factors are independent cell cycle events. Studies with vascular smooth muscle cells and endothelial cells

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Inhibition of rat cervical epithelial cell growth by heparin and its reversal by EGF

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12 December 2020