Activation of the murine EP3 receptor for PGE2 inhibits cAMP production and promotes platelet aggregation (Q28359873)

2

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K Athirakul

3

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K Coggins

4

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J D McNeish

5

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S Austin

6

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L K Parise

7

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G A FitzGerald

8

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T M Coffman

9

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B H Koller

Journal of Clinical Investigation

10

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language of work or name

English

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publication date

March 2001

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published in

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volume

107

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issue

5

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page(s)

603-10

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cites work

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Differential expression of prostaglandin E2 and interleukin-6 in occlusive and aneurysmal aortic disease

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On the multiplicity of platelet prostaglandin receptors I. Evaluation of competitive antagonism by aggregometry

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Arterial walls generate from prostaglandin endoperoxides a substance (prostaglandin X) which relaxes strips of mesenteric and coeliac ateries and inhibits platelet aggregation

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Effect of Prostaglandin E2 and Aspirin on the Secondary Aggregation of Human Platelets

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Arachidonic acid is preferentially metabolized by cyclooxygenase-2 to prostacyclin and prostaglandin E2

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Possible role of a cAMP-dependent phosphorylation in the calcium release mediated by inositol 1,4,5-trisphosphate in human platelet membrane vesicles

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Prostaglandin E2 potentiates platelet aggregation by priming protein kinase C

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Calcium-dependent stimulation of platelet aggregation by PGE2

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cAMP is an important messenger for ADP-induced platelet aggregation

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Concordant induction of prostaglandin E2 synthase with cyclooxygenase-2 leads to preferred production of prostaglandin E2 over thromboxane and prostaglandin D2 in lipopolysaccharide-stimulated rat peritoneal macrophages

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Relationship between cAMP and Ca2+ fluxes in human platelet membranes

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Distribution of prostaglandin IP and EP receptor subtypes and isoforms in platelets and human umbilical artery smooth muscle cells

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Interactions between prostaglandin E2 and inhibitors of platelet aggregation which act through cyclic AMP

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12 December 2020