Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice (Q28362795)

1

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G Esposito

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L Mao

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J R Hagaman

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J E Fox

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H A Rockman

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N Maeda

Journal of Clinical Investigation

8

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language of work or name

English

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publication date

April 2001

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published in

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volume

107

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issue

8

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page(s)

975-84

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cites work

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Cardiac overexpression of a G(q) inhibitor blocks induction of extracellular signal-regulated kinase and c-Jun NH(2)-terminal kinase activity in in vivo pressure overload

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Functional deletion mutation of the 5'-flanking region of type A human natriuretic peptide receptor gene and its association with essential hypertension and left ventricular hypertrophy in the Japanese

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Effect of long-term treatment with beta-blocker on cardiac hypertrophy in SHR

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Cardioreparative effects of lisinopril in rats with genetic hypertension and left ventricular hypertrophy

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Molecular characterization of the stretch-induced adaptation of cultured cardiac cells. An in vitro model of load-induced cardiac hypertrophy

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Effects of long-term enalapril therapy on cardiac structure and function in patients with left ventricular dysfunction. Results of the SOLVD echocardiography substudy

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Renin-angiotensin system stimulates cardiac and renal disorders in Tsukuba hypertensive mice

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Apoptosis during regression of cardiac hypertrophy in spontaneously hypertensive rats. Temporal regulation and spatial heterogeneity

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Extracellular signal-regulated protein kinase activation is required for the anti-hypertrophic effect of atrial natriuretic factor in neonatal rat ventricular myocytes

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The Mitogen-activated Protein Kinase Kinase MEK1 Stimulates a Pattern of Gene Expression Typical of the Hypertrophic Phenotype in Rat Ventricular Cardiomyocytes

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