Beryllium-stimulated production of tumor necrosis factor-alpha by a mouse hybrid macrophage cell line (Q28376166)

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L A Kittle

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L S Newman

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P A Campbell

Function and activation of NF-kappa B in the immune system

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language of work or name

English

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publication date

7 March 2000

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published in

Toxicology

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volume

143

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issue

3

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page(s)

235-47

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cites work

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Alveolar macrophages from patients with beryllium disease and sarcoidosis express increased levels of mRNA for tumor necrosis factor-alpha and interleukin-6 but not interleukin-1 beta

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Action of Beryllium on Cells cultivated in vitro; Effect on Mitosis

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Beryllium toxicity. The selective inhibition of casein kinase 1.

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Signal transduction in LPS-activated aged and young monocytes.

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LPS-binding proteins and receptors

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Expansions of T-cell subsets expressing particular T-cell receptor variable regions in chronic beryllium disease

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Beryllium disease. The relation of pulmonary pathology to clinical course and prognosis based on a study of 130 cases from the U.S. beryllium case registry

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Tumor necrosis factor alpha gene expression in human monocytic THP-1 cells exposed to beryllium

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5 Cloning of hormone genes from a mixture of cDNA molecules

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Immunologic specificity of lymphocyte cell lines from dogs exposed to beryllium oxide

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The effect of beryllium on the growth of human lung fibroblasts

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Pulmonary epithelial cell injury and alveolar-capillary permeability in berylliosis

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The pulmonary toxicity of beryllium

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Immunology, genetics, and epidemiology of beryllium disease

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The natural history of beryllium sensitization and chronic beryllium disease

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Binding of beryllium to nuclear acidic proteins

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Maintenance of alveolitis in patients with chronic beryllium disease by beryllium-specific helper T cells

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Structure and function of lipopolysaccharide binding protein

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Metal-induced infidelity during DNA synthesis

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Specific G1-S phase cell cycle block by beryllium as demonstrated by cytofluorometric analysis

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Cytokine production by bronchoalveolar lavage cells in chronic beryllium disease

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Beryllium-stimulated release of tumor necrosis factor-alpha, interleukin-6, and their soluble receptors in chronic beryllium disease

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Effects of beryllium on deoxyribonucleic acid-synthesizing enzymes in regenerating rat liver

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CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

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Lipopolysaccharide induction of the tumor necrosis factor-alpha promoter in human monocytic cells. Regulation by Egr-1, c-Jun, and NF-kappaB transcription factors

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