Fibroblast engraftment in the decellularized mouse lung occurs via a β1-integrin-dependent, FAK-dependent pathway that is mediated by ERK and opposed by AKT (Q28397455)

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English	Fibroblast engraftment in the decellularized mouse lung occurs via a β1-integrin-dependent, FAK-dependent pathway that is mediated by ERK and opposed by AKT	scientific article

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scholarly article

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Fibroblast engraftment in the decellularized mouse lung occurs via a β1-integrin-dependent, FAK-dependent pathway that is mediated by ERK and opposed by AKT (English)

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author name string

Huanxing Sun

1

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Elizabeth Calle

2

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Xiaosong Chen

3

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Aditi Mathur

4

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Yangyang Zhu

5

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Julio Mendez

6

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Liping Zhao

7

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Laura Niklason

8

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Xueyan Peng

9

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Hong Peng

10

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Erica L Herzog

11

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language of work or name

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publication date

15 March 2014

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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306

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6

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L463-75

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Phosphatidylinositol-3-Kinase/Akt regulates bleomycin-induced fibroblast proliferation and collagen production

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Focal adhesion kinase is upstream of phosphatidylinositol 3-kinase/Akt in regulating fibroblast survival in response to contraction of type I collagen matrices via a beta 1 integrin viability signaling pathway

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Integrins: bidirectional, allosteric signaling machines

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beta 1 integrin regulates fibroblast viability during collagen matrix contraction through a phosphatidylinositol 3-kinase/Akt/protein kinase B signaling pathway

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Identifiers

10.1152/AJPLUNG.00100.2013

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