TGF-β regulates DNA methyltransferase expression in prostate cancer, correlates with aggressive capabilities, and predicts disease recurrence (Q28477306)

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English	TGF-β regulates DNA methyltransferase expression in prostate cancer, correlates with aggressive capabilities, and predicts disease recurrence	scientific article

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scholarly article

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TGF-β regulates DNA methyltransferase expression in prostate cancer, correlates with aggressive capabilities, and predicts disease recurrence (English)

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prostate cancer

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based on heuristic

inferred from title

author name string

Qiang Zhang

1

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Lin Chen

2

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Brian T Helfand

3

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Thomas L Jang

4

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Vidit Sharma

5

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James Kozlowski

6

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Timothy Michael Kuzel

7

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Lihua J Zhu

8

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Ximing J Yang

9

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Borko Javonovic

10

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Yinglu Guo

11

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Scott Lonning

12

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Jay Harper

13

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Beverly A Teicher

14

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Charles Brendler

15

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Nengwang Yu

16

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William J Catalona

17

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Chung Lee

18

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language of work or name

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publication date

2011

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6

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9

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e25168

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copyright license

Creative Commons Attribution 4.0 International

30 September 2011

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April 2022 Public Data File from Crossref

copyright status

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TGF-beta activates Erk MAP kinase signalling through direct phosphorylation of ShcA

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19 March 2017

The DNA methyltransferases of mammals

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7 April 2017

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Insensitivity to transforming growth factor-beta results from promoter methylation of cognate receptors in human prostate cancer cells (LNCaP).

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DNA methyltransferase and demethylase in human prostate cancer

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https://pubmed.ncbi.nlm.nih.gov/21980391

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inferred from PubMed ID database lookup

On the mechanism of inhibition of DNA-cytosine methyltransferases by cytosine analogs

1 reference

https://pubmed.ncbi.nlm.nih.gov/21980391

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inferred from PubMed ID database lookup

Kinetic and catalytic mechanism of HhaI methyltransferase

1 reference

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based on heuristic

inferred from PubMed ID database lookup

Genetic change in transforming growth factor beta (TGF-beta) receptor type I gene correlates with insensitivity to TGF-beta 1 in human prostate cancer cells

1 reference

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0025168

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2011PLoSO...625168Z

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OpenCitations bibliographic resource ID

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

ResearchGate publication ID

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