The increased expression of integrin α6 (ITGA6) enhances drug resistance in EVI1(high) leukemia (Q28479144)

2

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Yusuke Saito

3

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Emi Ichihara

4

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Kazuhiro Morishita

Creative Commons Attribution 4.0 International

5

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language of work or name

English

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publication date

2012

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published in

PLOS One

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volume

7

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issue

1

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page(s)

e30706

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copyright license

start time

25 January 2012

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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Acetylation of lysine 564 adjacent to the C-terminal binding protein-binding motif in EVI1 is crucial for transcriptional activation of GATA2.

29 September 2017

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Combination therapy of an anticancer drug with the FNIII14 peptide of fibronectin effectively overcomes cell adhesion-mediated drug resistance of acute myelogenous leukemia

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Laminin-5 stimulates hepatocellular carcinoma growth through a different function of alpha6beta4 and alpha3beta1 integrins.

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Deletion of alpha4 integrins from adult hematopoietic cells reveals roles in homeostasis, regeneration, and homing.

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Identification of a common ecotropic viral integration site, Evi-1, in the DNA of AKXD murine myeloid tumors

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29 September 2017

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High EVI1 levels predict adverse outcome in acute myeloid leukemia: prevalence of EVI1 overexpression and chromosome 3q26 abnormalities underestimated.

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CD52 as a molecular target for immunotherapy to treat acute myeloid leukemia with high EVI1 expression

21 January 2018

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Molecular pathways in bone marrow homing: dominant role of alpha(4)beta(1) over beta(2)-integrins and selectins

21 January 2018

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Identification of a gene expression signature associated with pediatric AML prognosis

21 January 2018

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PubMed

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inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Comment on " 'Stemness': transcriptional profiling of embryonic and adult stem cells" and "a stem cell molecular signature"

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PubMed

https://pubmed.ncbi.nlm.nih.gov/22295105