Differential adaptation of Candida albicans in vivo modulates immune recognition by dectin-1 (Q28486281)

GOA release 2020-03-11

1 reference

Simon Vautier

2

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Mohlopheni Marakalala

Mohlopheni J Marakalala

1

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Hector M. Mora-montes

Hector M Mora-Montes

7

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Carol A Munro

Carol A Munro

13

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Donna M MacCallum

Donna M Maccallum

11

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Alistair J P Brown

Alistair J P Brown

16

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Neil A. R. Gow

14

Neil A R Gow

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Mihai Netea

9

Mihai G Netea

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Hopke Alex

1 reference

ORCID Public Data File 2021

author name string

Joanna Potrykus

3

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Louise A Walker

4

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Kelly M Shepardson

5

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Alex Hopke

6

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Ann Kerrigan

8

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Graeme I Murray

10

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Robert Wheeler

12

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Robert A Cramer

15

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Gordon D Brown

Creative Commons Attribution 4.0 International

17

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language of work or name

English

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publication date

2013

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published in

PLOS Pathogens

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volume

9

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issue

4

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page(s)

e1003315

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copyright license

start time

18 April 2013

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Dectin-1 isoforms contribute to distinct Th1/Th17 cell activation in mucosal candidiasis

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PPARγ controls Dectin-1 expression required for host antifungal defense against Candida albicans.

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Susceptibility to Coccidioides species in C57BL/6 mice is associated with expression of a truncated splice variant of Dectin-1 (Clec7a).

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Stimulation of chitin synthesis rescues Candida albicans from echinocandins.

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Differential high-affinity interaction of dectin-1 with natural or synthetic glucans is dependent upon primary structure and is influenced by polymer chain length and side-chain branching

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In vivo and ex vivo comparative transcriptional profiling of invasive and non-invasive Candida albicans isolates identifies genes associated with tissue invasion.

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ACandida albicansstrain with high MIC for caspofungin and noFKS1mutations exhibits a high chitin content and mutations in two chitinase genes

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12 December 2020

Candida albicans strain-dependent modulation of pro-inflammatory cytokine release by in vitro oral and vaginal mucosal models

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Toll-like receptor 4 defective mice carrying point or null mutations do not show increased susceptibility to Candida albicans in a model of hematogenously disseminated infection

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The GPI-anchored Gas and Crh families are fungal antigens

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10.1371/JOURNAL.PPAT.1003315

Identifiers

DOI

1 reference

1 reference

1 reference

PubMed publication ID

23637604

1 reference