Intracellular localisation of Fv1 (Q28508770)

Mechanism of restriction of murine leukemia viruses varies between different strains of Fv-1n mice

GOA release 2020-03-11

author name string

Melvyn W. Yap

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1

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24 January 2017

Jonathan P. Stoye

series ordinal

2

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24 January 2017

language of work or name

English

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publication date

1 March 2003

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24 January 2017

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24 January 2017

307

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24 January 2017

1

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24 January 2017

76–89

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24 January 2017

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Positional cloning of the mouse retrovirus restriction gene Fv1.

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Identification of the regions of Fv1 necessary for murine leukemia virus restriction.

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Use of a transient assay for studying the genetic determinants of Fv1 restriction

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Organization of organelles and membrane traffic by microtubules

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Infectious entry by amphotropic as well as ecotropic murine leukemia viruses occurs through an endocytic pathway

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Single amino acid changes in the murine leukemia virus capsid protein gene define the target of Fv1 resistance

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Action of cytochalasin D on cells of established lines. II. Cortex and microfilaments

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Analysis of mitochondrial morphology and function with novel fixable fluorescent stains

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HIV-1 Gag protein associates with F-actin present in microfilaments

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A transient three-plasmid expression system for the production of high titer retroviral vectors

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Rapid transport of internalized P-selectin to late endosomes and the TGN: roles in regulating cell surface expression and recycling to secretory granules

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A proline-rich region and nearby cysteine residues target XLalphas to the Golgi complex region

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Segregation of transferrin to a mildly acidic (pH 6.5) para-Golgi compartment in the recycling pathway

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Cloning of a new murine endogenous retrovirus, MuERV-L, with strong similarity to the human HERV-L element and with a gag coding sequence closely related to the Fv1 restriction gene

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Genetic analysis of the major homology region of the Rous sarcoma virus Gag protein

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A Mus dunni cell line that lacks sequences closely related to endogenous murine leukemia viruses and can be infected by ectropic, amphotropic, xenotropic, and mink cell focus-forming viruses

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Role of the major homology region of human immunodeficiency virus type 1 in virion morphogenesis.

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10.1016/S0042-6822(02)00053-3

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

513304

513304

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference